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Lipoprotein Lipase and Chylomicronemia Syndrome.
Lipoprotein Lipase and Chylomicronemia Syndrome.
Author
Prof. Hayk S. Arakelyan. Full Professor in Medicine,
Doctor of Medical Sciences, Ph.D , Grand Ph.D .
Senior Expert of Interactive Clinical Pharmacology , Drug Safety,
Treatment Tactics, General Medicine and Clinical Research.
“Time and health are two
precious assets that we don't
recognize and appreciate
until they have been
depleted .
“ “ Denis Waitley”
Introduction.
Chylomicronemia syndrome is a disorder in which the body does not break down fats (lipids) correctly. This causes fat particles called chylomicrons to build up in the blood. The disorder is passed down through families.
Causes.
Chylomicronemia syndrome can occur due to a rare genetic disorder in which a protein (enzyme) called lipoprotein lipase (LpL) is broken or missing. LpL is normally found in fat and muscle. It helps break down certain lipids. When LpL is missing or broken, fat particles called chylomicrons build up in the blood. This buildup is called chylomicronemia. Defects in apolipoprotein CII and apolipoprotein AV can cause the syndrome as well. It is more likely to occur when people who are predisposed to have high triglycerides (such as those who have familial combined hyperlipidemia or familial hypertriglyceridemia) develop diabetes, obesity or are exposed to certain medicines.
Symptoms.
The five most commonly reported symptoms in a survey of FCS patients were: Lipemia Retinalis, a condition in which the retinal veins of the eyes appear “milky”
-Memory Loss or ‘foggy-headedness.
-Depression or a sense of isolation.
-Severe pain in the abdomen often including back pain.
-Acute or chronic pancreatitis.
-Xanthomas, fatty deposits in the skin
Patients with FCS may also experience: -Vomiting or diarrhea.
-Blood that, when drawn, appears “milky”.
-Extremely high levels of triglycerides.
-Numbness in feet or legs.
The Risk of Pancreatitis. Pancreatitis can cause debilitating, life-threatening pain and is often more severe in patients with extremely high triglyceride levels.17 Bouts of pancreatitis typically begin with extreme nausea and vomiting, sweating, and pain in the upper abdomen.18 The pain can radiate to the back and ribcage and has been described by those who experience it and their caretakers as incapacitating, intolerable, "like someone stabbing me with a knife,"21 or "like someone took a power drill to the side of your rib cage. In a recent study of patients with acute pancreatitis who also had extremely high tryglyceride levels (≥1000 mg/dL).
-Differential Diagnosis of Chylomicronemia Syndrome.
- Other Diseases and Chylomicronemia Syndrome.
-Food and Chylomicronemia Syndrome.
-Drugs and Chylomicronemia Syndrome.
To be continued ....
If you have any questions concerning “ Lipoprotein Lipase and Chylomicronemia Syndrome.”, interactive clinical pharmacology , or any other questions, please inform me .
Prof. Hayk S. Arakelyan
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Text
Lipoprotein Lipase and Chylomicronemia Syndrome.
Author
Prof. Hayk S. Arakelyan. Full Professor in Medicine,
Doctor of Medical Sciences, Ph.D , Grand Ph.D .
Senior Expert of Interactive Clinical Pharmacology , Drug Safety,
Treatment Tactics, General Medicine and Clinical Research.
“Time and health are two
precious assets that we don't
recognize and appreciate
until they have been
depleted .
“ “ Denis Waitley”
Introduction.
Chylomicronemia syndrome is a disorder in which the body does not break down fats (lipids) correctly. This causes fat particles called chylomicrons to build up in the blood. The disorder is passed down through families.
Causes.
Chylomicronemia syndrome can occur due to a rare genetic disorder in which a protein (enzyme) called lipoprotein lipase (LpL) is broken or missing. LpL is normally found in fat and muscle. It helps break down certain lipids. When LpL is missing or broken, fat particles called chylomicrons build up in the blood. This buildup is called chylomicronemia. Defects in apolipoprotein CII and apolipoprotein AV can cause the syndrome as well. It is more likely to occur when people who are predisposed to have high triglycerides (such as those who have familial combined hyperlipidemia or familial hypertriglyceridemia) develop diabetes, obesity or are exposed to certain medicines.
Symptoms.
The five most commonly reported symptoms in a survey of FCS patients were: Lipemia Retinalis, a condition in which the retinal veins of the eyes appear “milky”
-Memory Loss or ‘foggy-headedness.
-Depression or a sense of isolation.
-Severe pain in the abdomen often including back pain.
-Acute or chronic pancreatitis.
-Xanthomas, fatty deposits in the skin
Patients with FCS may also experience: -Vomiting or diarrhea.
-Blood that, when drawn, appears “milky”.
-Extremely high levels of triglycerides.
-Numbness in feet or legs.
The Risk of Pancreatitis. Pancreatitis can cause debilitating, life-threatening pain and is often more severe in patients with extremely high triglyceride levels.17 Bouts of pancreatitis typically begin with extreme nausea and vomiting, sweating, and pain in the upper abdomen.18 The pain can radiate to the back and ribcage and has been described by those who experience it and their caretakers as incapacitating, intolerable, "like someone stabbing me with a knife,"21 or "like someone took a power drill to the side of your rib cage. In a recent study of patients with acute pancreatitis who also had extremely high tryglyceride levels (≥1000 mg/dL).
-Differential Diagnosis of Chylomicronemia Syndrome.
- Other Diseases and Chylomicronemia Syndrome.
-Food and Chylomicronemia Syndrome.
-Drugs and Chylomicronemia Syndrome.
To be continued ....
If you have any questions concerning “ Lipoprotein Lipase and Chylomicronemia Syndrome.”, interactive clinical pharmacology , or any other questions, please inform me .
Prof. Hayk S. Arakelyan
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Group Emotions and Spring .
Continued of Spring and Health- 1. Headache
Author
Prof. Hayk S. Arakelyan. Full Professor in Medicine,
Doctor of Medical Sciences, Ph.D , Grand Ph.D .
Senior Expert of Interactive Clinical Pharmacology , Drug Safety,
Treatment Tactics, General Medicine and Clinical Research.
“When spring came, even the false spring, there were no problems except where to be happiest. The only thing that could spoil a day was people and if you could keep from making engagements, each day had no limits. People were always the limiters of happiness except for the very few that were as good as spring itself.”
“ Ernest Hemingway,( A Moveable Feast)”
Introduction.
“True wisdom comes to each of us when we realize how little we understand about life, ourselves, and the world around us.”
“Socrates”
For a brief period, days and nights around the world each last close to 12 hours (day and night are not exactly equal, as the term “equinox” is meant to imply). Then, as the Earth continues its path around the sun, days become shorter and nights lengthen, with the change becoming more pronounced in the higher latitudes, but remaining nonexistent at the equator.This change in the amount of light is a signal to humans, animals, plants and, before the light bulb, people, of changing seasons. For some creatures living at high latitudes, it can have a profound effect on their biology, particularly on reproduction, which must be carefully timed.Earth's multiple motions — spinning on its axis and orbiting the sun — are behind everything from day and night to the changing seasons.The Earth's axis is tilted at 23.5 degrees, which makes the Northern Hemispheres point more directly at the sun the sun half the year, and the Southern Hemisphere do the same the other half. Group emotion refers to the moods, emotions and dispositional affects of a group of people. It can be seen as either an emotional entity influencing individual members' emotional states (top down) or the sum of the individuals' emotional states (bottom up). Collective emotions are at the heart of any society and become evident in gatherings, crowds, or responses to widely salient events. However, they remain poorly understood and conceptualized in scientific terms. Here, we provide first steps towards a theory of collective emotions. We first review accounts of the social and cultural embeddedness of emotion that contribute to understanding collective emotions from three broad perspectives: face-to-face encounters, culture and shared knowledge, and identification with a social collective. In discussing their strengths and shortcomings and highlighting areas of conceptual overlap, we translate these views into a number of bottom–up mechanisms that explain collective emotion elicitation on the levels of social cognition, expressive behavior, and social practices.
To be continued ....
-Spring and Allergy.
-Spring and Sinusitis.
-Spring and Migraine.
-Spring and Cardiovascular Disease.
-Spring and Pulmonary Diseases.
-Spring and Pain Syndrome.
-Spring and Love.
-Spring and Brain Performance.
-Spring and Creativity.
-Spring and Ulcer.
-Spring and Gastrointestinal Diseases.
If you have any questions concerning “Group Emotions and Spring .”, interactive clinical pharmacology , or any other questions, please inform me .
Prof. Hayk S. Arakelyan
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Dangerous Beauties- 2. Honey from poisonous flowers. Continued of Dangerous Beauties- 1 Room Plants.
Author
Prof. Hayk S. Arakelyan. Full Professor in Medicine,
Doctor of Medical Sciences, Ph.D , Grand Ph.D .
Senior Expert of Interactive Clinical Pharmacology , Drug Safety,
Treatment Tactics, General Medicine and Clinical Research.
“Flowers always make people
better, happier, and more
helpful; they are sunshine,
food and medicine for the
soul.”
“Luther Burbank”
Introduction.
Dangerous Beauties- 2. Honey from poisonous flowers.
Continued of Dangerous Beauties- 1 Room Plants.
Author
Prof. Hayk S. Arakelyan. Full Professor in Medicine,
Doctor of Medical Sciences, Ph.D , Grand Ph.D .
Senior Expert of Interactive Clinical Pharmacology , Drug Safety,
Treatment Tactics, General Medicine and Clinical Research.
“Flowers always make people
better, happier, and more
helpful; they are sunshine,
food and medicine for the
soul.”
“Luther Burbank”
Introduction.
Plants that produce nectar that is poisonous to either honey bees or humans. Plants can produce chemicals in sap, pollen, nectar or honeydew that are toxic to honey bees and humans. Some plants, such as the linden, are usually considered an excellent source of nectar, although under certain specific stress conditions, they have been shown to poison bees. These "specific stress conditions" seem to occur repeatedly in most cases of poisoning and they affect the dose of poison the bees receive. When environmental conditions, especially soil moisture, reduce other sources of nectar, the bee is forced to forage from the toxic source because it is the only food available. Under "normal" moisture, other sources are available that dilute the amount of toxic substance to a level below the threshold of a toxic response. The dose is the amount or quantity of chemical present. It is important to understand the concept of dose, because the quantity of a chemical toxin from a poisonous plant at a low dose may be a valuable medicine, while at higher dose the same chemical can be deadly. A substance that is toxic to one organism may not be toxic to another. If honey produced from one plant nectar is toxic to humans, it cannot be assumed that the nectar or honey from this plant is also toxic to bees. The converse is also possible, that nectar or honey from a toxic plant may be toxic to bees but not to humans. Some plants that have been reported as poisonous are listed below. Summer Titi - Cyrilla racemiflora – is toxic to honey bees and can cause the condition called “purple brood”. Rhododendron from the heath family (Ericaceae) is poisonous to bees and humans. It contains an andromedotoxin. Mountain Laurel (Kalmia latifolia) also contains an andromedotoxin which can poison humans. Rhododendron poisoning.
Grayanotoxin Poisoning: Mad Honey Disease
.
Grayanotoxins are a group of closelyrelated toxins found in rhododendrons and other plants of the family Ericaceae. They can be found in honey made from their nectar and cause a very rare poisonous reaction called grayanotoxin poisoning,honey intoxication, or rhododendron poisoning.
Rhododendron
Rhododendrons are grown for their spectacular flowers, usually borne in spring. Some also have young leaves and stems covered in a striking dense woolly covering (indumentum) and some - the deciduous rhododendrons or azaleas - have good autumn colour. Rhododendron is a large genus of flowering plants. It includes 1,024 species of woody plants in the heath family (Ericaceae). Some are evergreen, and some are deciduous. They are found mainly in Asia.
Most of them have quite showy flowers. What gardeners know as Azalea is in fact a kind of rhododendron. Many people have rhododendrons in their garden or flower pot. It is the national flower of Nepal. One rhododendron, Rhododendron ponticum, has become an Invasive species in North America.
-Other plants that make poisonous honey.
-Intoxications from poisonous honey.
- trees toxic to bees.
- poisonous nectar.
- Oleander honey.
To be continued ....
If you have any questions concerning “ Dangerous Beauties- 2. Honey from poisonous flowers.”, interactive clinical pharmacology , or any other questions, please inform me .
Prof. Hayk S. Arakelyan
Grayanotoxins are a group of closelyrelated toxins found in rhododendrons and other plants of the family Ericaceae. They can be found in honey made from their nectar and cause a very rare poisonous reaction called grayanotoxin poisoning,honey intoxication, or rhododendron poisoning. Rhododendron
Rhododendrons are grown for their spectacular flowers, usually borne in spring. Some also have young leaves and stems covered in a striking dense woolly covering (indumentum) and some - the deciduous rhododendrons or azaleas - have good autumn colour. Rhododendron is a large genus of flowering plants. It includes 1,024 species of woody plants in the heath family (Ericaceae). Some are evergreen, and some are deciduous. They are found mainly in Asia.
Most of them have quite showy flowers. What gardeners know as Azalea is in fact a kind of rhododendron. Many people have rhododendrons in their garden or flower pot. It is the national flower of Nepal. One rhododendron, Rhododendron ponticum, has become an Invasive species in North America.
-Other plants that make poisonous honey.
-Intoxications from poisonous honey.
- trees toxic to bees.
- poisonous nectar.
- Oleander honey.
To be continued ....
If you have any questions concerning “ Dangerous Beauties- 2. Honey from poisonous flowers.”, interactive clinical pharmacology , or any other questions, please inform me .
Prof. Hayk S. Arakelyan
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Body Dysmorphic Disorder.
Continued of Dangerous Ingredients In Beauty Products and Cosmetics
Author
Prof. Hayk S. Arakelyan. Full Professor in Medicine,
Doctor of Medical Sciences, Ph.D , Grand Ph.D .
Senior Expert of Interactive Clinical Pharmacology , Drug Safety,
Treatment Tactics, General Medicine and Clinical Research.
“The human body is the best
picture of the human soul.”
“ Ludwig Wittgenstein”
Introduction.
Body dysmorphic disorder (BDD) is a distinct mental disorder in which a person is preoccupied with an imagined physical defect or a minor defect that others often cannot see. As a result, people with this disorder see themselves as "ugly" and often avoid social exposure or turn to plastic surgery to try to improve their appearance. BDD shares some features with eating disorders and obsessive-compulsive disorder.
Symptoms.
Body dysmorphic disorder typically starts in the early teenage years and it affects both males and females. An obsession that body build is too small or not muscular enough (muscle dysmorphia) occurs almost exclusively in males. Insight about body dysmorphic disorder varies. You may recognize that your beliefs about your perceived flaws may not be true, or think that they probably are true, or be absolutely convinced that they're true. Signs and symptoms of body dysmorphic disorder include: -Being extremely preoccupied with a perceived flaw in appearance that to others can't be seen or appears minor.
-Strong belief that you have a defect in your appearance that makes you ugly or deformed.
-Belief that others take special notice of your appearance in a negative way or mock you.
-Engaging in behaviors aimed at fixing or hiding the perceived flaw that are difficult to resist or control, such as frequently checking the mirror, grooming or skin picking.
-Attempting to hide perceived flaws with styling, makeup or clothes.
-Constantly comparing your appearance with others.
-Always seeking reassurance about your appearance from others
-Having perfectionist tendencies.
-Seeking frequent cosmetic procedures with little satisfaction.
-Avoiding social situations.
-Being so preoccupied with appearance that it causes major distress or problems in your social life, work, school or other areas of functioning.
-Risk factors of Body dysmorphic disorder (BDD).
-Prevention of Body dysmorphic disorder.
- Complications of Body dysmorphic disorder.
-Diagnosis and Treatment Perspectives of Body dysmorphic disorder.
To be continued ....
If you have any questions concerning “ Body Dysmorphic Disorder.”, interactive clinical pharmacology , or any other questions, please inform me .
Prof. Hayk S. Arakelyan
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Text
Schnitzler Syndrome and IgM .
Author
Prof. Hayk S. Arakelyan. Full Professor in Medicine,
Doctor of Medical Sciences, Ph.D , Grand Ph.D .
Senior Expert of Interactive Clinical Pharmacology , Drug Safety,
Treatment Tactics, General Medicine and Clinical Research.
“Do not give in too much to feelings. A overly sensitive heart is an unhappy possession on this shaky earth.”
“Johann Wolfgang von Goethe”
Introduction.
The disease is named after the French dermatologist Liliane Schnitzler who first described this syndrome in 1972. Schnitzler syndrome is characterized by chronic, nonpruritic urticaria in association with recurrent fever, bone pain, arthralgia or arthritis, and a monoclonal gammopathy, most often of the immunoglobulin M (IgM) subtype. Approximately 10-15% of patients eventually develop a lymphoproliferative disorder, such as lymphoplasmacytic lymphoma, Waldenström macroglobulinemia, or IgM myeloma.
Signs and symptoms.
Schnitzler syndrome is characterized by the following signs and symptoms: recurrent hives, mostly on the torso and limbs, often with recurring fever, joint pain, bone pain, muscle pain, headache, fatigue, and loss of weight.
Diagnosis.
Immunoglobulin M (IgM) or rarely IgG monoclonal gammopathy: Detected with serum immunoelectrophoresis; occurs in all patients; in 51% of cases, however, serum protein electrophoresis may not detect it. Blood tests show a high concentration of specific gamma-globulins (monoclonal gammopathy) of the IgM type. It almost always has light chains of the κ-type. A variant in which IgG is raised has been described, which appears to be one-tenth as common. The immunoglobulins may show up in the urine as Bence Jones proteins. Signs of inflammation are often present: these include an increased white blood cell count (leukocytosis) and a raised erythrocyte sedimentation rate and C-reactive protein. There can be anemia of chronic disease. Abnormal lymphoid proliferation: 20% of bone marrow biopsy samples, with nonspecific polyclonal lymphocytic and plasmacytic infiltrates.
-Management and Treatment Perspectives.
-Autoimmunisation and Schnizler Syndrome.
-
Pathophysiology
.
-Epidemiology.
- IgM, IGG and Schnizler Syndrome.
- IgM, IGG and other Diseases.
- IgM and IgG class antibodies and Allergy Types.
To be continued ....
If you have any questions concerning “Schnitzler Syndrome and IgM .”, interactive clinical pharmacology , or any other questions, please inform me.
Prof. Hayk S. Arakelyan
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Leptin and Food Addiction.
Leptin and Food Addiction.
Continued of Chinese Kitchen Syndrome.
Author
Prof. Hayk S. Arakelyan. Full Professor in Medicine,
Doctor of Medical Sciences, Ph.D , Grand Ph.D .
Senior Expert of Interactive Clinical Pharmacology , Drug Safety,
Treatment Tactics, General Medicine and Clinical Research.
“Let food be thy medicine, thy medicine shall be thy food.”
“Hippocrates”
Introduction.
Leptin is a hormone predominantly made by adipose cells that helps to regulate energy balance by inhibiting hunger. Leptin is opposed by the actions of the hormone ghrelin, the "hunger hormone". Both hormones act on receptors in the arcuate nucleus of the hypothalamus to regulate appetite to achieve energy homeostasis.
Leptin and Ghrelin.
Leptin and ghrelin are two hormones that have been recognized to have a major influence on energy balance. Leptin is a mediator of long-term regulation of energy balance, suppressing food intake and thereby inducing weight loss. Ghrelin on the other hand is a fast-acting hormone, seemingly playing a role in meal initiation. As a growing number of people suffer from obesity, understanding the mechanisms by which various hormones and neurotransmitters have influence on energy balance has been a subject of intensive research.
The influence of leptin on the dopamine system and implications for ingestive behavior. Food intake is regulated by many factors, including sensory information, metabolic hormones, and the state of hunger. In modern humans, the drive to eat has proven to be incompatible with the excess food supply present in industrialized societies. Rates of obesity are increasing worldwide. The natural drive to eat, combined with a surplus of readily available food, are together partly responsible for this modern epidemic. In order to understand the source of this problem, we need to better define the molecular and neural mechanisms by which the brain regulates food intake, and why it is often difficult to control food ingestion in times of excess. The discovery that peripheral metabolic hormones, normally studied for their activity in the hypothalamus, act directly on dopamine neurons of brain dopamine centers to modulate feeding behavior, has provided new mechanisms for behavioral control. Here, recent findings are reviewed and with an emphasis on the potential implications as well as open questions that remain. Glutamate and Leptin. Leptin is a protein secreted by adipocytes that is important in regulating appetite and adiposity. Recent studies have suggested the presence of leptin receptors in the arcuate nucleus of the hypothalamus (ANH). Neonatal administration of monosodium glutamate (MSG) damages the ANH, resulting in obesity and neuroendocrine dysfunction. Dopamine D2 receptors in addiction-like reward dysfunction and compulsive eating. development of obesity was coupled with emergence of a progressively worsening deficit in neural reward responses. Similar changes in reward homeostasis induced by cocaine or heroin are considered to be crucial in triggering the transition from casual to compulsive drug-taking. Accordingly, we detected compulsive-like feeding behavior. Administration of leptin has been shown to modulate behaviors that are dependent upon the mesolimbic dopamine circuit. For example, leptin administration alters intracranial self-stimulation, suggesting an interaction with dopamine circuits that are thought to underlie the behavior. Other data have demonstrated that leptin attenuates the increased propensity to heroin relapse caused by food restriction (
12
) and can modulate conditioned place preference for sucrose or high fat food. These data suggest that leptin can modify reward based behavior and that this may occur via alteration in function of dopamine pathways . While this interaction with dopamine pathways could occur downstream from leptin’s effects on the hypothalamus, recent data also suggests a direct action of leptin on dopamine centers of the brain.
-Dopamine and food intake.
-Mutant Leptins.
- Genetic Aspects and location of genes .
-Leptin , ghrelin body weight in humans and Diseases.
-Dopamine, the nucleus accumbens, and hedonics.
-
Regulation of glutamate receptor trafficking by leptin
. -
Glutamate and GABA in Appetite Regulation
.
-Food Addiction and Drugs.
-Food Addiction and Alcohol.
-Food Addiction and Smoking.
-Food Addiction and Diseases.
-Food Addiction and Brain Performance.
To be continued ....
If you have any questions concerning “ Leptin and Food Addiction.”, interactive clinical pharmacology , or any other questions, please inform me .
Prof. Hayk S. Arakelyan
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Text
Oxidative Stress and Health Continued of Antioxidants.-Anti Aging Secrets-3
Authors
1. Hayk S. Arakelyan.
Full Professor in Medicine,
Doctor of Medical Sciences, Ph.D , Grand Ph.D .
Senior Expert of Interactive Clinical Pharmacology , Drug Safety,
Treatment Tactics, General Medicine and Clinical Research.
2. Hsiu-Hsien Tsai
Medical school, Taipei Medical University Changhua County, Taiwan
“Every human being is the author of his own health or disease.”
“Buddha”
Introduction.
Oxidative stress leads to many pathophysiological conditions in the body. Some of these include neurodegenerative diseases such as Parkinson's disease and Alzheimer's disease, gene mutations and cancers, chronic fatigue syndrome, fragile X syndrome, heart and blood vessel disorders, atherosclerosis, heart failure, heart attack and inflammatory diseases. Every cell that utilizes enzymes and oxygen to perform functions is exposed to oxygen free radical reactions that have the potential to cause serious damage to the cell. Antioxidants are molecules present in cells that prevent these reactions by donating an electron to the free radicals without becoming destabilized themselves. An imbalance between oxidants and antioxidants is the underlying basis of oxidative stress.
Free Radicals.
A free radicals is an oxygen containing molecule that has one or more unpaired electrons, making it highly reactive with other molecules. Oxygen by-products are relatively unreactive but some of these can undergo metabolism within the biological system to give rise to these highly reactive oxidants. Not all reactive oxygen species are harmful to the body. Some of them are useful in killing invading pathogens or microbes. However, free radicals can chemically interact with cell components such as DNA, protein or lipid and steal their electrons in order to become stabilized. This, in turn, destabilizes the cell component molecules which then seek and steal an electron from another molecule, therefore triggering a large chain of free radical reactions.
-Oxidative Stress and Cancer.
-Oxidative Stress and Other Diseases.
-Oxidative Stress and Food.
-Oxidative Stress and Drugs.
-Oxidative Stress and Immune Defense.
-Oxidative Stress and Antioxidants.
-
Oxidative stress: its role in air pollution and adverse health effects
.
To be continued ....
If you have any questions concerning ’ Oxidative Stress and Health .’, interactive clinical pharmacology , or any other questions, please inform us.
Prof . Hayk S. Arakelyan
Hsiu-Hsien Tsai
Medical school, Taipei Medical University Changhua County, Taiwan
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Text
Chinese Kitchen Syndrome.
Author
Prof. Hayk S. Arakelyan. Full Professor in Medicine,
Doctor of Medical Sciences, Ph.D , Grand Ph.D .
Senior Expert of Interactive Clinical Pharmacology , Drug Safety,
Treatment Tactics, General Medicine and Clinical Research.
“Let food be thy medicine, thy medicine shall be thy food.”
“Hippocrates”
Introduction.
Chinese restaurant syndrome is a set of symptoms that some people have after eating Chinese food. A food additive called monosodium glutamate (MSG) has been blamed for the condition. Reports of serious reactions to Chinese food first appeared in 1968. At that time, MSG was thought to be the cause of these symptoms. Chinese food and soups contain monosodium glutamate (MSG) as the main addictive ingredient. Individual may suffer from headache, giddiness, sweating, abdominal pain, and urticaria within a few hours of consumption of MSG. Angioedema may be delayed up to 8–16 h after the consumption of MSG and it may persist for 24 h.This delayed life-threatening effect in the form of angioedema makes diagnosis difficult.
Glutamate flavoring. Glutamate flavoring is a generic name for
flavor-enhancing compounds
based on
glutamic acid
and its
salts
(glutamates). These compounds provide an
umami
(savory) taste to food. Glutamic acid and glutamates are natural constituent of many
fermented
or aged foods, including
soy sauce
,
fermented bean paste
, and
cheese
, and also occur in
hydrolyzed protein
such as
yeast extract
. The
sodium
salt of glutamic acid,
monosodium glutamate
(MSG) is manufactured in a large scale and widely used in the
food industry
. Only the L-glutamate
enantiomer
has flavor-enhancing properties. Manufactured monosodium glutamate contains over 99.6% of the naturally-predominant L-glutamate form, which is a higher proportion of L-glutamate than found in the free glutamate ions of fermented naturally-occurring foods. Fermented products such as
soy sauce
,
steak sauce
, and
Worcestershire sauce
have levels of glutamate similar to foods with added monosodium glutamate. Cognitive and biochemical effects of monosodium glutamate and aspartame, administered individually and in combination on the cognitive behavior and biochemical parameters like neurotransmitters and oxidative stress indices in the brain tissue. Symptoms. Reports of serious reactions to Chinese food first appeared in 1968. At that time, MSG was thought to be the cause of these symptoms. There have been many studies since then that have failed to show a connection between MSG and the symptoms some people describe. Symptoms may include
headache
, throbbing of the head,
dizziness
, lightheadedness, a feeling of facial pressure, tightness of the jaw, burning or tingling sensations over parts of the body,
chest pain
, and back pain. MSG continues to be used in some meals. However people are particularly sensitive to food additives. MSG is chemically similar to one of the brain's most important chemicals, glutamate.Symptoms often include a headache, skin flushing, and sweating. A food additive called monosodium glutamate (MSG) is often blamed for the symptoms some people experience after eating this food. However, there’s minimal scientific evidence showing a link between MSG and these symptoms in humans. The U.S. Food and Drug Administration (FDA) considers MSG to be a safe ingredient, and most people can eat foods that contain MSG without experiencing any problems. However, a small percentage of people have short-term adverse reactions to the food additive. Due to the controversy, many restaurants now advertise that they don’t add MSG to their foods. Less commonly, people experience severe, potentially life-threatening symptoms that are similar to those of allergic reactions. -Treatment and Profylactic Perspectives. - Glutamate flavoring and brain chemicals. - Glutamate flavoring and Other Diseases. - Glutamate flavoring and Food Interactions. -Glutamate flavoring and Drug Interactions. -Glutamate flavoring and Alcohol Interactions. - MSG allergy. -Japanese restaurant syndrome.
To be continued ....
If you have any questions concerning “Chinese Kitchen Syndrome”, interactive clinical pharmacology , or any other questions, please inform me.
Prof. Hayk S. Arakelyan
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Text
Overactive Bladder Syndrome
Author
Prof. Hayk S. Arakelyan. Full Professor in Medicine,
Doctor of Medical Sciences, Ph.D , Grand Ph.D .
Senior Expert of Interactive Clinical Pharmacology , Drug Safety,
Treatment Tactics, General Medicine and Clinical Research.
“Good health and good sense are two of life's greatest
blessings.”
“Publilius Syrus”
Introduction.
An overactive bladder is a condition resulting from the sudden, involuntary contraction of the muscle in the wall of the bladder.An overactive bladder causes an uncontrollable and unstoppable urge to pass urine and the frequent need to urinate both during the daytime and night, even though the bladder may only contain a small amount of urine. It is sometimes referred to as small bladder syndrome. The condition affects around 15% of adults, women affected more frequently than men. The incidence also increases as you get older.
Causes and symptoms of an overactive bladder.
The exact cause of an overactive bladder is a mystery. However, several factors are known to contribute to the involuntary contraction of the bladder muscle, improper bladder function, and other symptoms associated with an overactive bladder. Some nervous system abnormalities that can cause an overactive bladder include: Parkinson’s disease, Multiple Sclerosis, Spinal cord injury, Stroke, Dementia etc. Other causes of overactive bladder can include: Urinary Tract Infection, Bladder stones, Trauma to the pelvis or abdomen (eg a car accident), Nerve damage or trauma caused by surgery or certain therapies, Enlarged prostate etc. Overactive bladder symptoms can include: Sudden and overwhelming urge to urinate — This symptom can come on very quickly and without any warning. Frequency — This is the need to urinate more often than usual — sometimes more than eight times within a 24 hour period. Often the need to urinate is ever-present whether fluid intake is limited or not. Urge incontinence — This happens when you suddenly leak urine after feeling an urgent and immediate need to go to the toilet. There’s also a feeling of not being able to reach the toilet fast enough. Nocturia — This occurs when an overactive bladder wakes you to use the toilet several times during the night to the point where your sleep cycle is disrupted.
-treatments for an overactive bladder.
-Lifestyle changes during overactive bladder.
- Risk factors of overactive bladder.
- Complications of overactive bladder.
To be continued ....
If you have any questions concerning “ Overactive Bladder Syndrome ”, interactive clinical pharmacology , or any other questions, please inform me.
Prof. Hayk S. Arakelyan
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Driving phobia. - Vehophobia in Forensic Medicine .
Driving phobia. - Vehophobia in Forensic Medicine .
Continued of Strange Phobias-2.
Author
Prof. Hayk S. Arakelyan. Full Professor in Medicine,
Doctor of Medical Sciences, Ph.D , Grand Ph.D .
Senior Expert of Interactive Clinical Pharmacology , Drug Safety,
Treatment Tactics, General Medicine and Clinical Research.
“Courage is resistance to
fear, mastery of fear, not
absence of fear.”
“Mark Twain”
Introduction.
Driving phobia is a form of agoraphobia, literally defined as is the fear of open spaces. But it's not the fear of open spaces that scares people, it's the fear of loss of control. People with a driving phobia fear being trapped in a traffic jam and unable to escape if they experience a panic attack, likewise, they also fear passing out, losing control of the vehicle, throwing up or getting into an accident. For many people, driving next to big trucks can be very nerve racking, as can be merging on the freeway or driving in the fast lane.
Driving Anxiety.
Driving anxiety is a very common form of anxiety that can range in severity from a hesitation to drive, where anxiety is always present, all the way up to a total refusal to drive at all, in which case it becomes driving phobia. A phobia is a fear that is paralyzing but irrational. Driving phobia is one of the most common phobias.
Driving phobia tips.
- The basics: Avoid driving on an empty stomach. Pay attention to how you feel after eating certain foods, especially those high in sugar or simple carbs (bread, pastries, soft drinks). Drinking alcohol the night before can also trigger blood sugar imbalances. Also, if you are driving while sleep deprived, you are asking for trouble. Start by taking care of yourself.
- Consider car pooling: If you are engaged in conversation you are less liable to think anxious thoughts. You also have to drive half as much. Think this one over carefully, as some people are more distracted while conversing while driving.
- Affirmations: Hand write, in script, some positive affirmation about your ability to drive calm, comfortable and relaxed. For example "I'm calm, comfortable and relaxed while driving and enjoying listening to music (the radio, audio books, etc.)" Read them right before you go to bed and right after you wake up. Say them out loud and imagine yourself driving while feeling calm and relaxed. Don't underestimate the power of this simple exercise.
Causes and symptoms of vehophobia.
The first and most common cause of a fear of driving is traffic accidents. The second most common form is driving phobia as a specific phobia. Because driving does involve some danger and the possibility of a collision, there does exist some fear or caution in many rational people. People with a fear of driving may experience trembling, sweating, accelerated pulse, loss of sense of reality, and thoughts of losing control while driving, even in situations that are reasonably safe. This fear will cause many to avoid driving, create excuses to not drive, or even refuse to get a driver's license for years.
- Panic Disorder.
- Past negative experiences and driving phobia.
- Fear of Fatalities and driving phobia.
- Vehophobia and forensic medicines.
Treatment perspectives of driving phobia.
To be continued ....
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Food and Drug Induced Reward Deficiency Syndrome .
Food and Drug Induced Reward Deficiency Syndrome .
Author
Prof. Hayk S. Arakelyan. Full Professor in Medicine,
Doctor of Medical Sciences, Ph.D , Grand Ph.D .
Senior Expert of Interactive Clinical Pharmacology , Drug Safety,
Treatment Tactics, General Medicine and Clinical Research.
“The only way to keep your
health is to eat what you
don't want, drink what you
don't like, and do what you'd
rather not.”
“Mark Twain”
Introduction.
Reward Deficiency Syndrome (RDS) a name for a relative failure of the dopaminergic system which plays a major part in brainreward mechanisms. The syndrome, whichhas been linked to dysfunction of the D2 dopamine receptors, includes various conditions, such as food, drug and alcohol abuse, obesity, pathological gambling and attention deficit hyperactivitydisorder, in which the subject seems to be unusually concerned to achieve reward. RDS is a dysfunction of the reward system observed in some individuals taking dopaminergic medications for an extended length of time. It is characterized by self-control problems such as addiction to medication, gambling, or sexual behavior.
Dopamine dysregulation syndrome. The most common symptom is craving for dopaminergic medication. However other behavioral symptoms can appear independently of craving or co-occur with it. Craving is an intense impulse of the subject to obtain medication even in the absence of symptoms that indicate its intake. To fulfill this need the person will self-administer extra doses. When self-administration is not possible, aggressive outbursts or the use of strategies such as symptom simulation or bribery to access additional medication can also appear.
Hypomania
, manifesting with feelings of
euphoria
, omnipotence, or grandiosity, are prone to appear in those moments when medication effects are maximum;
dysphoria
, characterized by sadness, psychomotor slowing,
fatigue
or
apathy
are typical with dopamine replacement therapy (DRT) withdrawal.
Pleasure deprivation within the reward centers of the brain.
Those afflicted with RDS are believed to experience a significant degree of pleasure depravation within the reward center of the brain. The severity of the condition is subject to variation based on the individual; other traits can play a role. Based on addiction research, dependency of various drugs such as alcohol, opiates, and stimulants all seem to have a similar biological basis in the brain. Addictions and reward-deficiency behaviors seem to stem from within the limbic system, specifically in two regions. These regions are known as the “nucleus accumbens” and the “globus pallidus” – and express reward among those with various addictions.
Symptoms of Reward Deficiency Syndrome:
Those with reward-deficiency syndrome tend to experience a variety of symptoms. These symptoms cannot necessarily be used to “diagnose” the condition because there is no specific diagnostic criteria. However, those who are aware of the condition may realize that they may be dealing with a case of reward-deficiency. Addiction(s): People with RDS may develop addictions as a way to feel engaged with life. Using drugs or gambling may make someone with this condition feel some degree of pleasure and/or thrill. This allows them to feel more “normal” and experience the same level of reward that others get from more mundane activities. Therefore certain addictions such as using drugs or gambling may become the focal point of the individual’s life. Lack of anticipation: Most people get excited while anticipating an event such as the first day on a new job, first day at school, going on a vacation, or a wedding. For most people, the excitement leading up to these events is significant. Those with reward-deficiency may not feel any excitement leading up to these major events. Essentially the excitement that normal people experience is heavily muted for those with RDS. Lack of pleasure: Due to the significantly reduced number of D2 receptors, many individuals don’t get a thrill from any activity. Certain events such as going on a big date, taking a vacation, or getting a good grade on a test may not make a person with RDS feel good. Some individuals with this condition may experience very little pleasure from “rewarding” activities, while others may experience none at all.
- Self-Medicating with Reward-Deficiency Syndrome.
-Drugs and Reward-Deficiency Syndrome.
-Alcohol and Reward-Deficiency Syndrome.
-Sugars and Reward-Deficiency Syndrome.
- Gambling and Reward-Deficiency Syndrome.
-Sex and Reward-Deficiency Syndrome.
-Food and Reward-Deficiency Syndrome.
- Reward-Deficiency Syndrome Treatment.
To be continued ....
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Prof. Hayk S. Arakelyan
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Pathological lyar Syndrome – Mythomania.
Continued of Antisocial Personality Disorder in Forensic Medicine.
Author
Prof. Hayk S. Arakelyan. Full Professor in Medicine,
Doctor of Medical Sciences, Ph.D , Grand Ph.D .
Senior Expert of Interactive Clinical Pharmacology , Drug Safety,
Treatment Tactics, General Medicine and Clinical Research.
“Liars are always most disposed to swear.”
“ Vittorio Alfieri ”
Introduction.
Pathological lying (also called pseudologia fantastica and mythomania) is a behavior of habitual or compulsive lying. Pathological lying is also known a compulsive lying. When pathological liars are forced to speak the truth during a conversation they feel uncomfortable and often react in an animated manner.
Identifying pathological liar.
Signs and Symptoms of Pathological liars.
1. Pathological liars know just one thing… lies. Any situation they are in they lie. These guys use their verbal skills to good effect in different situations.
2. Pathological liars love to create a comfort zone for themselves. In a bid to get there, they cook up tales and stories with manic conviction.
3. Pathological liars love attention and may go any length with their lies to get it. These guys are aware of the fact that lies and deception get them a fair deal of attention and so they seek refuge in lies all the time. A pathological liar takes pride in grandiose acts achieved through fake sentiments and lies.
4. Pathological liars have a keen sense of perception and are seldom caught lying. If they sense they are going to be caught, they weave another story to cover up their lies.
5. Pathological liars suffer from low self esteem which is why they use lies as a means to feel better about.
6. Pathological liars live in a world of fantasy. They love to create a false world among people they know and get a high once people fall for their lies. The only time a pathological liar’s ego is hurt is when lies are not believed.
7. Pathological liars may exhibit behavioral changes with different people, especially people they have not known for a long time.
8. Pathological liars rely on convincing fake narratives to engage the person or persons they are talking to. Their stories involve a theatrical element to evoke a response. Often stories about exploits and bravery are fake to the bone. Every time they meet different people the concept and centrifugal element of their stories remain same while all other aspects change.
9. Pathological liars use camouflage and deception in personality traits displayed with different people. These guys can mould themselves to be submissive or dominant when need arise in accordance with the situation encountered.
10. A pathological liar loves to have the last word. These guys love to prove their point even if they know they are wrong and the other person is right.
- Treatment perspectives for pathological liars.
- Pathological lyar Syndrome and Mental Disorders.
-Pathological lyar Syndrome and Other Diseases.
-Pathological lyar Syndrome and Alcohol.
-Pathological lyar Syndrome and Drugs.
-Pathological lyar Syndrome and Love.
To be continued ....
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Prof. Hayk S. Arakelyan
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Smoking and Skin.
Smoking and Skin.
Continued of Smoking and Stress.
Author
Prof. Hayk S. Arakelyan. Full Professor in Medicine,
Doctor of Medical Sciences, Ph.D , Grand Ph.D .
Senior Expert of Interactive Clinical Pharmacology , Drug Safety,
Treatment Tactics, General Medicine and Clinical Research.
“The only way to keep your
health is to eat what you
don't want, drink what you
don't like, and do what you'd
rather not.”
“Mark Twain”
Introduction.
Smoking’s immediate effects on the skin. It may lead to chronic skin diseases. One of the most reasonable explanations as to how smoking affects the skin is through decreasing the oxygen in the blood and constricting the blood vessels. Also, studies find that the damaging effects of smoking on the skin are increased when combined with UV rays that you get from direct sunlight exposure.
Smoking and Skin Ageing.
Smoking causes changes in the composition of the skin, causing premature ageing. Based on scientific studies, the skin loses its elasticity and acquires small red lines due to the dilation of blood vessels. The more you smoke, the more your skin ages rapidly. The nicotine content in a cigarette causes tiny blood vessels at the surface of the skin to tighten. With the tightening of the vessels comes a lowering in the blood supply to the area. Nicotine also already reduces the amount of oxygen in your blood, and with the oxygen further lowered, the skin becomes damaged. Other nutrients also get forced away from the skin during this time. If the blood supply cannot effectively deliver what the skin needs to remain healthy, damage is expedited. Skin becomes not only vulnerable to what smoking does, it also leaves the skin unprotected from other common sources of irritation and destruction, like environmental pollution and natural aging. In addition, many of the chemicals in modern cigarettes can do damage to cells that help to retain a youthful appearance. Collagen and elastin are there to promote healthy skin with plenty of stretch to it. When those are damaged, the skin can start to sag or wrinkle instead of holding it’s shape and bouncing back from being stretched or folded. Nicotine also works as a diuretic in the body, often leading to dehydration. This dehydration extends to the skin, where dryness makes skin easier to damage.
Smoking and Skin discoloration.
Skin discoloration is one of the most apparent consequences of smoking. It either turns grayish or orange, losing its youthful glow.
Smoking causes wrinkles.
Smoking can speed up the normal aging process of your skin, contributing to wrinkles. These skin changes may occur after only 10 years of smoking. The more cigarettes you smoke and the longer you smoke, the more skin wrinkling you're likely to have — even though the early skin damage from smoking may be hard for you to see initially. Aside from age, smoking is the strongest predictor of facial wrinkling in men and women. Smoking doesn't cause wrinkles only on your face. Smoking is also associated with increased wrinkling and skin damage on other parts of your body, including your inner arms.
-Smoking and skin infections.
-Smoking and psoriasis.
-Smoking and Lupus.
-Smoking and other skin diseases.
-Smoking and Mood.
-Smoking and Depression.
-Smoking and Creativity.
To be continued ....
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Prof. Hayk S. Arakelyan
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Strange Skin Parasites-1. Fleas
Strange Skin Parasites-1. Fleas
Author
Prof. Hayk S. Arakelyan. Full Professor in Medicine,
Doctor of Medical Sciences, Ph.D , Grand Ph.D .
Senior Expert of Interactive Clinical Pharmacology , Drug Safety,
Treatment Tactics, General Medicine and Clinical Research.
“To keep the body in good
health is a duty... otherwise
we shall not be able to keep
our mind strong and clear.”
“ Buddha”
Introduction.
Fleas are small flightless insects. A small wingless jumping insect which feeds on the blood of mammals and birds. It sometimes transmits diseases through its bite, including plague and myxomatosis.
Symptoms. Fleas are parasites that feed on blood. Fleas can live on any warm-blooded animal, but seem to prefer to live on humans, cats, dogs, opossums, rats and other rodents. They can also be found on shoes, pant legs, or blankets. Fleas are best known for spreading the Bubonic Plague. They also transmit the bacterial disease murine typhus to humans. Their saliva is an allergen that can cause allergic reactions in pets and humans. Fleas can also transfer tapeworms and cause anemia in pets. Flea bites cause painful, itchy red bumps. Fleabites exhibit several common symptoms. They are very itchy, and the skin around each bite may become sore or painful. And you may experience hives or develop a rash near the site of a bite. Additionally, excessive scratching can further damage the skin and a secondary bacterial infection can develop. Fleas will bite petless humans, too. If you don’t have a pet, your fleabites could be coming from your yard or another person’s animal. Fleas prefer tall grass and shaded areas near decks, woodpiles, or storage buildings. These wingless insects are known for their leaping abilities. They also reproduce quickly, making their numbers a force to be reckoned with. To prevent a possible infestation of these parasites, be aware of signs that they're present in your home. Cats and dogs increase scratching and fur-chewing when they have fleas. They also develop telltale black specks known as "flea dirt" in their fur. On your skin, fleas will leave clusters or lines of fleabites. These areas will have itchy dark red bumps ringed in pink.
- Fleas and Allergy.
- Fleas and other Diseases.
-Fleas and Food.
-Fleas and Laser Therapy.
- Profilaxis of Fleas.
- Treatment Perspectives of Fleas.
- Types of Fleas.
-The Dangers of Fleas.
To be continued ....
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Prof. Hayk S. Arakelyan
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Fifth Disease,Skin and Parvovirus
Author
Prof. Hayk S. Arakelyan. Full Professor in Medicine,
Doctor of Medical Sciences, Ph.D , Grand Ph.D .
Senior Expert of Interactive Clinical Pharmacology , Drug Safety,
Treatment Tactics, General Medicine and Clinical Research.
“Good health and good sense
are two of life's greatest
blessings.”
“Publilius Syrus”
Introduction.
Fifth disease is a viral infection caused by parvovirus B19. Fifth disease spreads easily, through saliva and mucus. You can get it when an infected person coughs or sneezes. Frequently washing your hands might help prevent getting the virus. The Latin name for the disease is erythema infectiosum, meaning infectious redness. It is alsocalled the "slapped cheek disease" because, when the bright red rash first appears on the cheeks,
Causes and symptoms.
Eythema infectiosum is caused by the parvovirus B19. It involves a low-grade fever, tiredness, and a rash over the body and, notably, on the cheeks. The symptoms of fifth disease include:
The main symptoms.- slightly elevated body temperature, flu-like symptoms, fatigue, sore throat, runny or stuffy nose, rash. A rash appears in three stages. A blotchy red rash may appear on the cheeks. Red papules emerge. These group together within a few hours to form red, slightly swollen, warm plaques. They do not affect the nose or mouth. After about 4 days, a net of red marks may appear on the arms and trunk. These form into a lacy pattern. The third stage is recurrent. It is invisible, but exposure to sunlight or heat may cause it to appear. After about 3 weeks, the rash will normally disappear, but it can last from 1 to 6 weeks. The rash may be itchy, but it is rarely painful. As it tends to appear towards the end of the illness, it may be mistaken for a drug-related rash or another disease.
Less common symptoms are:
nausea, diarrhea, or both,abdominal pain, arthralgia, or joint pain, normally only in adults.
Adults may experience pain and soreness in their joints, especially the hands, wrists, knees, and ankles. Joint pain can last from 2 weeks to over a year.
Rarely, an adult may experience neurological or cardiovascular problems.
Transmission and Diagnosis.
Parvovirus B19 spreads between humans through the air, saliva, or as a result of close contact.The most common form of transmission is through sneezing or coughing, and sometimes hand-to-hand contact.It can spread rapidly in places where many people congregate, such as schools, kindergartens, and nurseries. Rarely, transmission may occur through blood products. Signs and symptoms of erythema infectiosum tend to be easy to detect, so diagnosis is normally straightforward. It is sometimes confused with scarlet fever, but the rash is different.
-Differential Diagnosis of Fifth Disease.
- Treatment Perspectives of Fifth Disease.
To be continued ....
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Prof. Hayk S. Arakelyan
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M oon and Health.
Continued of Moon and Human Health-1. Selena Medicine.
Author
Prof. Hayk S. Arakelyan. Full Professor in Medicine,
Doctor of Medical Sciences, Ph.D , Grand Ph.D .
Senior Expert of Interactive Clinical Pharmacology , Drug Safety,
Treatment Tactics, General Medicine and Clinical Research.
“Three things cannot be long
hidden: the sun, the moon,
and the truth.”
“Buddha”
Introduction.
The moon's gravitational pull is undoubtedly powerful. Just look at the tides. So it's logical to assume that it will influence the fluids flowing through our body, like blood, mucus, and brain chemicals.
Human Sanogenesis and Moon.
Our bodies extensively use elements such as calcium, potassium and sodium ions for transmitting electrical signals between the brain and the nerves. Large quantities of ions can therefore immensely affect our health because they interfere with our own internal communications system, at a cellular level. The term lunar effect refers to real or imaginary correlations between specific stages of the roughly 29.5-day lunar cycle and behavior and physiological changes in living beings on Earth, including humans. In some cases the purported effect may depend on external cues, such as the amount of moonlight.
Brain. Much like the Moon is responsible for the ebb and flow of tides, since our brains are a significant source of water,researchers hypothesize the Moon’s gravitational pull could similarly have an effect on your brain, causing erratic behavior.
Heart.
The Moon’s cycles have an affect on your heart. Research found that when exercising, your heart is at its peak performance during a full and new Moon.
Menstrual Cycle. A woman’s average menstrual cycle is 28 days, which is quite similar to the 29+ day lunar cycle. This timing may be more than coincidence according to Chinese researchers who discovered that almost 30 percent of all the women monitored ovulated at the full Moon and menstruated during the new Moon. In some cultures there is even a name for this phenomenon, called White Moon Cycle, that essentially mirrors the fertility of the Earth which is said to be most fertile under full moonlight.
Increased Birthrate.
Researchers discovered something interesting: there was a significant increase in the number of births when the gravitation of the Moon to the Earth was most powerful. While researchers admit they are still unclear on exactly how or why this relationship exists, they say it might help healthcare professionals and pregnant women better prepare.
Increased Injuries.
Since the full Moon can affect our behavior in certain ways, it should come as no surprise that we are more prone to accidents or illness during this time, according to some.
Sleep.
people longer to fall asleep during a full moon. In addition, the rate of brain activity that indicates a restful, deep sleep dropped by 30 percent, and, overall, they slept for 20 minutes less. "The lunar cycle seems to influence human sleep, even when one does not see the moon and is not aware of the actual moon phase,"
Heart attack.
A study found a significant reduction in heart attacks during the three days following a new moon.
- Human behavior and Moon. - Mental Illness and Moon. - Moon phase and Diseases. - Epilepsy and Moon. -Moon and Human Hormones.
-Moon and Love.
-Moon and Creativity,
To be continued ....
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