#Nephrolithiasis Pain Diagnosis Medicine

[lexingtonrenalcare]

Lexington Renal Care

Provides comprehensive care for patients who are facing kidney disease, kidney transplant hospital in Lexington, KY transplants, dialysis, and hypertension.   Our patients are our highest priority. We understand that each the patient is unique and individual needs will be considered when deciding on treatment options.

[journalofclinical]

Vertebral Osteomyelitis Masquerading as Pulmonary Embolism

Clinical entities known to mimic the presentation of pulmonary embolism (PE) include pneumonia, asthma, bronchitis, chronic obstructive airway disease (COPD) flare, congestive heart failure, acute myocardial infarction, primary pulmonary artery sarcoma, and nephrolithiasis. Only two cases of vertebral osteomyelitis mimicking PE are found in the literature. We report a rare case of lumbar vertebral osteomyelitis presenting with pleuritic chest pain and shortness of breath, and therefore causing delayed diagnosis.

Read more about articles: https://lupinepublishers.com/clinical-community-medicine/fulltext/vertebral-osteomyelitis-masquerading-as-pulmonary-embolism.ID.000123.php

Read more Lupine Publishers Google Scholar articles:

https://scholar.google.com/citations?view_op=view_citation&hl=en&user=vfbCW-wAAAAJ&cstart=20&pagesize=80&citation_for_view=vfbCW-wAAAAJ:ruyezt5ZtCIC

[mcatmemoranda]

We have a pt who has rhabdomyolysis from taking a statin and being on the floor for 3 days. She's old, so she had muscle breakdown. The attending is Dr. Giraldo, who is really nice and awesome. He asked me what urine test you use to diagnose rhabdo and I totally forgot about myoglobinuria so I said I wasn't aware. He told me to look it up. Then after we were rounding for a bit, my brain remembered myoglobinuria! So I told him. But now I want to look up the details of diagnosing rhabdo so I can present to him tomorrow. But we also have a pt with CKD who may have kidney stones and the attending and the resident didn't realize that you can diagnose a kidney stone with a non-contrast CT. I remembered learning that last year during my emergency medicine rotation. They had ordered a KUB because they thought you would need to use contrast for the CT and so they didn't want to get the CT because the contrast would hurt the pt's kidney. But you don't use contrast to diagnose nephrolithiasis with CT. So at least I remembered something! This is from UpToDate:

●The clinical manifestations of rhabdomyolysis include myalgias, weakness, red to brown urine due to myoglobinuria, and elevated serum muscle enzymes (including creatine kinase [CK]). The degree of myalgias and other symptoms varies widely, and some patients are asymptomatic. Fever, malaise, tachycardia, and gastrointestinal symptoms may be present. Muscle swelling may occur with rehydration.

This pt was actually tachycardic in the ED. So that tracks.

●The laboratory findings that characterize rhabdomyolysis include an acute elevation in the CK and other muscle enzymes and a decline in these values within three to five days of cessation of muscle injury. The other characteristic finding is the reddish-brown urine of myoglobinuria, but this finding is often absent because of the relative rapidity with which myoglobin is cleared. The serum CK is generally entirely or almost entirely of the MM or skeletal muscle fraction, although small amounts of the MB fraction may be present.

●Other manifestations include fluid and electrolyte abnormalities, many of which precede or occur in the absence of acute kidney injury, and hepatic injury. Hypovolemia, hyperkalemia, hyperphosphatemia, hypocalcemia, hyperuricemia, and metabolic acidoses may be seen. [I think the pt also had hypocalcemia, but it wasn't true hypocalcemia because the albumin was low, so her corrected Ca2+ was in the normal range based on the lab values at the hospital; she did have acidosis too I think]. Hyperkalemia may result in cardiac dysrhythmias. Later complications include acute kidney injury (AKI), hypercalcemia, compartment syndrome, and, rarely, disseminated intravascular coagulation.

●We diagnose rhabdomyolysis in a patient with an acute muscular illness or injury based upon a marked acute elevation in serum CK; the CK is typically at least five times the upper limit of normal and is frequently greater than 5000 international units/L. Key diagnostic laboratory studies include the creatine kinase and urinalysis, including dipstick and microscopic evaluation. Myoglobinuria (present in 50 to 75 percent of patients at the time of initial evaluation) results in a positive test for blood on the urine dipstick but without red blood cells on the microscopic examination of the urine. And for this pt, the UA showed a small amount of blood, so that could have been myoglobin in the urine, but we didn't order a microscopic analysis. She also has a UTI, so that could be from the UTI as well. Also, the other day Dr. Agarwal asked how long you treat UTIs. When in the hospital, you can treat with ceftriaxone until the pt has clinically improved.

●The differential diagnosis depends upon the combination of findings present. It includes myocardial infarction, other causes of red or brown urine, inflammatory myopathy, and local causes of pain, such as deep vein thrombosis or renal colic.

The characteristic triad of complaints in rhabdomyolysis is muscle pain, weakness, and dark urine. Additional symptoms that are more common in severely affected patients include malaise, fever, tachycardia, nausea and vomiting, and abdominal pain. Altered mental status may occur from the underlying etiology (eg, toxins, drugs, trauma, or electrolyte abnormalities).

The hallmark of rhabdomyolysis is an elevation in CK and other serum muscle enzymes. The other characteristic finding is the reddish-brown urine of myoglobinuria, but because this may be observed in only half of cases, its absence does not exclude the diagnosis. Routine lab tests, including complete blood count (CBC), erythrocyte sedimentation rate (ESR), and C-reactive protein (CRP), vary greatly depending on the underlying cause of rhabdomyolysis. Infections and crush injuries are associated with marked elevation of the acute phase reactants and peripheral white blood cell (WBC) count, while these markers of inflammation would likely be normal or only minimally raised in patients with other etiologies, such as drug-induced or electrolyte derangements.

Serum CK levels at presentation are usually at least five times the upper limit of normal, but range from approximately 1500 to over 100,000 international units/L. The mean peak CK reported for each of a variety of different causes and for patients with both single and multiple causes ranged from approximately 10,000 to 25,000 in the largest series; exceptions were the three patients with malignant hyperthermia, whose values averaged almost 60,000.

I googled the normal serum CK level:

In a healthy adult, the serum CK level varies with a number of factors (gender, race and activity), but normal range is 22 to 198 U/L (units per liter). Higher amounts of serum CK can indicate muscle damage due to chronic disease or acute muscle injury.

The CK is generally entirely or almost entirely of the MM or skeletal muscle fraction; a small proportion of the total CK may be from the MB or myocardial fraction. The presence of MB reflects the small amount found in skeletal muscle rather than the presence of myocardial disease. Elevations in serum aminotransferases are common and can cause confusion if attributed to liver disease. In one study, aspartate aminotransferase (AST) was elevated in 93.1 percent and alanine aminotransferase (ALT) in 75 percent of rhabdomyolysis cases in which the CK was greater than or equal to 1000 units/L. In only one instance was the ALT greater than the AST, although the AST declines faster than the ALT as the rhabdomyolysis resolves, such that the two may equalize after a few days.

The serum CK begins to rise within 2 to 12 hours following the onset of muscle injury and reaches its maximum within 24 to 72 hours. A decline is usually seen within three to five days of cessation of muscle injury. CK has a serum half-life of about 1.5 days and declines at a relatively constant rate of about 40 to 50 percent of the previous day's value. In patients whose CK does not decline as expected, continued muscle injury or the development of a compartment syndrome may be present.

Urine findings and myoglobinuria — Myoglobin, a heme-containing respiratory protein, is released from damaged muscle in parallel with CK. Myoglobin is a monomer that is not significantly protein-bound and is therefore rapidly excreted in the urine, often resulting in the production of red to brown urine. It appears in the urine when the plasma concentration exceeds 1.5 mg/dL. Visible changes in the urine only occur once urine levels exceed from about 100 to 300 mg/dL, although it can be detected by the urine (orthotolidine) dipstick at concentrations of only 0.5 to 1 mg/dL . Myoglobin has a half-life of only two to three hours, much shorter than that of CK. Because of its rapid excretion and metabolism to bilirubin, serum levels may return to normal within six to eight hours.

Thus, it is not unusual for CK levels to remain elevated in the absence of myoglobinuria. In rhabdomyolysis, myoglobin appears in the plasma before CK elevation occurs and disappears while CK is still elevated or rising. Therefore, there is no CK threshold for when myoglobin appears. As above, rhabdomyolysis does not occur unless CK is elevated five times or more above the upper limit of normal. Routine urine testing for myoglobin by urine dipstick evaluation may be negative in up to half of patients with rhabdomyolysis. Pigmenturia will be missed in rhabdomyolysis if the filtered load of myoglobin is insufficient or has largely resolved before the patient seeks medical attention due to its rapid clearance.

Both hemoglobin and myoglobin can be detected on the urine dipstick as "blood;" microscopic evaluation of the urine generally shows few red blood cells (RBC) (less than five per high-powered field) in patients with rhabdomyolysis whose positive test results from myoglobinuria. Such testing is not a reliable method for rapid detection of myoglobin if RBC are present or in patients with hemolysis due to its lack of specificity for myoglobin. Hemoglobin, the other heme pigment capable of producing pigmented urine, is much larger (a tetramer) than myoglobin and is protein-bound. As a result, much higher plasma concentrations are required before red to brown urine is seen, resulting in a change in plasma color.

Hypocalcemia, which can be extreme, occurs in the first few days because of entry into damaged myocytes and both deposition of calcium salts in damaged muscle and decreased bone responsiveness to parathyroid hormone. During the recovery phase, serum calcium levels return to normal and may rebound to significantly elevated levels due to the release of calcium from injured muscle, mild secondary hyperparathyroidism from the acute renal failure, and an increase in calcitriol (1,25-dihydroxyvitamin D).

Severe hyperuricemia may develop because of the release of purines from damaged muscle cells and from reduced urinary excretion if acute kidney injury occurs.

●Metabolic acidosis is common, and an increased anion gap may be present. Our pt did have an anion gap and I wondered why. I guess it's because there's more uric acid in the blood.

Acute kidney injury — Acute kidney injury (AKI, acute renal failure) is a common complication of rhabdomyolysis. The reported frequency of AKI ranges from 15 to over 50 percent. The risk of AKI is lower in patients with CK levels at admission less than 15 to 20,000 units/L; risk factors for AKI in patients with lower values include dehydration, sepsis, and acidosis. [Our pt had peed a lot and was on the floor for 2 to 3 days, so she was probably dehydrated, increasing her risk for AKI]. Volume depletion resulting in renal ischemia, tubular obstruction due to heme pigment casts, and tubular injury from free chelatable iron all contribute to the development of renal dysfunction. Reddish-gold pigmented casts are often observed in the urine sediment.

Compartment syndrome — A compartment syndrome exists when increased pressure in a closed anatomic space threatens the viability of the muscles and nerves within the compartment. Compartment syndrome is a potential complication of severe rhabdomyolysis that may develop after fluid resuscitation, with worsening edema of the limb and muscle. Lower extremity compartment syndrome can also be a cause of rhabdomyolysis, as may occur after tibial fractures.

Disseminated intravascular coagulation — Infrequently, severe rhabdomyolysis may be associated with the development of disseminated intravascular coagulation due to the release of thromboplastin and other prothrombotic substances from the damaged muscle.

EVALUATION AND DIAGNOSIS

Indications for diagnostic testing — Diagnostic testing should be performed in individuals with:

●Both myalgias and pigmenturia.

●Either myalgias or pigmenturia, with a history suggesting the presence or recent exposure to a potential cause or event.

●The absence of myalgias and pigmenturia in a clinical setting associated with increased risk for rhabdomyolysis, as symptoms may be vague or absent in up to 50 percent of patients. The diagnosis should be suspected following prolonged immobilization [like our pt who was on the floor for 2 to 3 days], in any stuporous or comatose patient, or in a patient who is otherwise unable to provide a medical history and has one or more of the following:

•Muscle tenderness

•Evidence of pressure necrosis of the skin

•Signs of multiple trauma or a crush injury

•Blood chemistry abnormalities suggesting the possibility of increased cell breakdown, such as hyperkalemia, hyperphosphatemia, and/or hypocalcemia

•Evidence of acute kidney injury

●Acute muscle weakness and marked elevation of creatine kinase (CK).

Diagnostic evaluation — We obtain the following key diagnostic laboratory studies:

●Creatine kinase – In addition to elevation of the CK, other muscle enzymes are typically elevated (eg, aldolase, aminotransferases, lactate dehydrogenase), but such testing is not usually necessary to make the diagnosis. However, elevations in aminotransferases or lactate dehydrogenase may suggest the need for CK testing if it has not been performed in a patient in whom such abnormalities may potentially be due to muscle injury rather than hepatic injury or another cause.

●Urinalysis, including dipstick and microscopic evaluation – Evidence of myoglobinuria should be sought by routine urine dipstick evaluation combined with microscopic examination. Testing of the unspun urine or the supernatant of the centrifuged urine will be positive for "heme" on dipstick if myoglobinuria is present, even if red to reddish brown urine is not evident macroscopically. The visual and microscopic examination of the sediment from a fresh urine specimen is required to exclude the presence of red blood cells (RBC) as the cause of positive testing; RBC in an older specimen may hemolyze over time, confounding the results.

In patients with persistent red to reddish-brown urine, myoglobinuria is suggested when the urine tests positive for heme by dipstick after centrifugation, while the plasma has a normal color and tests negative for heme.

Myoglobinuria lacks sensitivity as a test for rhabdomyolysis; it may be absent in 25 to 50 percent of patients with rhabdomyolysis due to the more rapid clearance of myoglobin, compared with CK, following muscle injury. Myoglobin also decreases rapidly in a similar fashion in patients with renal failure, suggesting a role for extrarenal metabolism and clearance in such patients.

We also obtain the following tests, which may help in prompt recognition of other potentially dangerous manifestations, in differential diagnosis, and in identifying the cause:

●Complete blood count, including differential and platelet count

●Blood urea nitrogen, creatinine, and routine electrolytes including potassium

●Calcium, phosphate, albumin, and uric acid

●Electrocardiography

Additional testing, such as evaluation of suspected metabolic myopathy or toxicology screening for drugs of abuse, depends upon the clinical context.

Diagnosis — We make the diagnosis of rhabdomyolysis in a patient with either an acute neuromuscular illness or dark urine without other symptoms, plus a marked acute elevation in serum creatine kinase (CK). The CK is typically at least five times the upper limit of normal, and is usually greater than 5000 international units/L. No absolute cut-off value for CK elevation can be defined, and the CK should be considered in the clinical context of the history and examination findings.

MANAGEMENT

The major issues in the treatment of patients with rhabdomyolysis include:

●Recognition and management of fluid and electrolyte abnormalities, which should be initiated regardless of renal function and which may prevent severe metabolic disturbances and acute kidney injury

●Identification of the specific causes and the use of appropriate countermeasures directed at the triggering events, including discontinuation of drugs or other toxins that may be etiologic factors

●Prompt recognition, evaluation, and treatment of compartment syndrome in patients in whom it is present

[faheemkhan882]

*Common Abbreviations*

> Rx = Treatment.

> Hx = History

> Dx = Diagnosis

> q = Every

> qd = Every day

> qod = Every other day

> qh = Every Hour

> S = without

> SS = On e half

> C = With

> SOS = If needed

> AC = Before Meals

> PC = After meals

> BID = Twice a Day

> TID = Thrice a Day

> QID = Four times a day

> OD = Once a Day

> BT = Bed Time

> hs = Bed Time

> BBF = Before Breakfast

> BD = Before Dinner

> Tw = Twice a week

> SQ = sub cutaneous

> IM = Intramuscular . .

> ID = Intradermal

> IV = Intravenous

> Q4H = (every 4 hours)

> QOD = (every other day)

> HS = (at bedtime)

> PRN = (as needed)

> PO or "per os" (by mouth)

> AC (before meals)

> PC = (after meals)

> Mg = (milligrams)

> Mcg/ug = (micrograms)

> G or Gm = (grams)

> 1TSF ( Teaspoon) = 5 ml

> 1 Tablespoonful =15ml

~ DDx=differential Diagnosis

Tx=Treatment

RTx=Radiotherapy

CTx=Chemotherapy

R/O=rule out

s.p=status post

PMH(x)=post medical history

Px=Prognosis

Ix=Indication

CIx=contraindication

Bx =biopsy

Cx=complication...

Knowledge About Blood.

1. Which is known as ‘River of Life’?

Answer: Blood

2. Blood circulation was discovered by?

Answer: William Harvey

3. The total blood volume in an adult?

Answer: 5-6 Litres

4. The pH value of Human blood?

Answer: 7.35-7.45

5. The normal blood cholesterol level?

Answer: 150-250 mg/100 ml

6. The fluid part of blood?

Answer: Plasma

7. Plasma protein fibrinogen has an active role in?

Answer: Clotting of blood.

8. Plasma protein globulins functions as?

Answer: Antibodies

9. Plasma proteins maintain the blood pH?

Answer: Albumins

10. Biconcave discs shaped blood cell?

Answer: RBC (Erythrocytes)

11. Non nucleated blood cell?

Answer: RBC (Erythrocytes)

12. Respiratory pigments present in RBC?

Answer: Haemoglobin

13. Red pigment present in RBC?

Answer: Haemoglobin

14. RBC produced in the?

Answer: Bone marrow

15. Iron containing pigment of Haemoglobin?

Answer: Haem

16. Protein containing pigment of Haemoglobin?

Answer: Globin

17. Graveyard of RBC?

Answer: Spleen

18. Blood bank in the body?

Answer: Spleen

19. Life span of RBC?

Answer: 120 Days

20. Total count is measured by an instrument known as?

Answer: Haemocytometer

21. A decrease in RBC count is known as?

Answer: Anemia

22. An increase in RBC count is known as?

Answer: Polycythemia

23. A high concentration of bilirubin in the blood causes?

Answer: Jaundice

24. The disease resistant blood cell?

Answer: WBC (leucocytes)

25. Which WBC is known as soldiers of the body?

Answer: Neutrophils

26. Largest WBC?

Answer: Monocyes

27. Smallest WBC?

Answer: Lymphocytes

28. Antibodies producing WBC?

Answer: Lymphocytes

29. Life span of WBC?

Answer: 10-15 days

30. Blood cell performs an important role in blood clotting?

Answer: Thrombocytes (Platelets)

31. Vessels is called?

Answer: Thrombus

32. Anticoagulant present in Blood?

Answer: Heparin

33. A hereditary bleeding disease?

Answer: Haemophilia

34. Bleeder’s disease?

Answer: Haemophilia

35. Christmas disease?

Answer: Haemophilia

36. A type of Anemia with sickle shaped RBC?

Answer: Sickle cell anemia

37. Viscosity of Blood?

Answer: 4.5 to 5.5

38. Instrument used to measure haemoglobin?

Answer: Haemoglobinometer

39. Who demonstrated blood groups?

Answer: Karl Landsteiner

40. Who demonstrated Rh factor?

Answer: Karl Landsteiner

41. Blood group which is called Universal donor?

Answer: O

42. Blood group which is called Universal recipient?

Answer: AB

43. Blood group is most common among the Asians?

Answer: B

MEDICAL TERMINOLOGY

Everybody should know the basic functioning of Human Body and its main parts in order to express and explain their ailment to the Doctor and at the same time one should be able to understand the diagnosis expressed by the Doctor in the medical terminology. For easy recognition of the Compounded Words used in the Medical Terminology for naming the disease, Suffixes are added to Prefixes. For this hereunder giving you a few such prefixes for your ready reference and understanding.

Prefix - Meaning

1. Adeno - Glandular

2. An - Not

3. Anti - Against

4. Aorto - Aorta

5. Artho - joint

6. Bleph - Eyelid

7. Broncho - Bronchi

8. Cardio - Heart

9. Cephal - Head

10. Cerebro - Brain

11. Cervico - Cervix

12. Cholecysto - Gall Bladder

13. Coli - Bowel

14. Colpo - Vagina

15. Entero - Intestine

16. Gastro - Stomach

17. Glosso - Tongue

18. Haema - Blood

19. Hepa - Liver

20. Hystero - Uterus

21. Laryngo - Larynx

22. Leuco - White

23. Metro - Uterus

24. Myelo - Spinal cord

25. Myo - Muscle

26. Nephro - Kidney

27. Neuro - Nerve

28. Odonto - Tooth

29. Orchido - Testis

30. Osteo - Bone

31. Oto - Ear

32. Pharyngo - Pharynx

33. Pio - Pus

34. Pneumo - Lung

35. Ren - Kidney

36. Rhin - Nose

37. Spleno - Spleen

38. Thyro - Thyroid Gland

39. Urethro - Urethra

40. Vesico – Bladder

Here are the suffixes used in Medical terminology. Check out!

Suffix - Meaning

1. -aemia : Blood

2. -algia : Pain

3. -derm : skin

4. -dynia : pain

5. -ectomy : removal

6. -Itis : inflammation

7. -lithiasis : Presence of Stone

8. -malacia : softening

9. -oma : tumour

10. -opia : eye

11. -osis : Condition,excess

12. -otomy : incision of

13. -phobia : fear

14. -plasty : surgery

15. -plegia : peralysis

16. -ptosis : falling

17. -rhoea : excessive discharge

18. -rhage : to burst forth

19. -rhythmia : rhythm.

20. -stasis : stoppage of movement

21. -sthenia : weakness

22. -stomy : outlet

23. -tomy : removal

24. -trophy : nourishment

25. -uria : urine

Compounded Words - Meaning

1. Anaemia - Deficiency of haemoglobin in the blood

2. Analgesic - Medicine which alleviates pain

3. Arthralgia - Pain in a joint

4. Cephalalgia - Headache

5. Nephralgia - Pain in the kidney

6. Neuralgia - Nerve pain

7. Myalgia - Muscle pain

8. Otalgia - Ear ache

9. Gastralgia - Pain in the stomach

10. Pyoderma - Skin infection with pus formation

11. Leucoderma -Defective skin pigmentaion

12. Hysterodynia - Pain in the uterus

13. Hysterectomy - Excision of the uterus

14. Nephrectomy - Excision of a kidney

15. Adenectomy - Excision of a gland

16. Cholecystectomy - Excision of gall bladder

17. Thyroidectomy - Excision of thyroid gland

18. Arthritis - Inflammation of a joint

19. Bronchitis - Inflammation of the bronchi

20. Carditis - Inflammation of the heart

21. Cervicitis - Inflammation of the cervix

22. Colitis - Inflammation of the colon

23. Colpitis - Inflammation of the vagina

24. Cystitis - Inflammation of the urinary bladder

25. Enteritis - Inflammation of the intestines

26. Gastritis - Inflammation of the stomach

27. Glossitis - Inflammation of the tongue

28. Hepatitis - Inflammation of the liver

29. Laryngitis - Inflammation of the larynx

30. Metritis - Inflammation of the uterus

31. Myelitis - Inflammation of the spinal cord

32. Nephritis - Inflammation of the kidney

33. Pharyngitis - Inflammation of the pharynx

34. Blepharitis -Inflammation of the eyelids

35. Cholelithiasis - Stone in the gall bladder

36. Nephrolithiasis - Stone in the kidney

37. Osteomalacia - Softening of bones through deficiency of calcium or D vitamin

38. Adenoma -Benign tumour of glandular tissue

39. Myoma - Tumour of muscle

40. Diplopia - Double vision

41. Thrombosis - Formation of a blood clot

[kidneyayurveda]

Which treatment is the best for the renal stone of 14mm size?

A kidney stone is the hard, crystalline mineral that usually formed within the kidney or urinary tract. The medical term of the kidney stone is Nephrolithiasis, and it is estimated that one in every 20 people develops the kidney at some point in their life. Usually, kidney stones formed when there is a decrease in the urine volume. These can be painful and also lead to other severe kidney-related problems.

Kidney stones can be as small as a grain of the sand and pass out of the body in your urine without causing any discomfort. But when the size of the kidney stones, you may experience extreme pain and problem in pea.

When to see the doctor?

Generally, the small kidney stones may pass out from the body through the urine without causing any symptoms. But the large kidney stones may cause symptoms that are mentioned below:

Severe pain in the back or side

Blood in the urine that may change the color of the urine

More urge to urinate

A burning sensation during urination

Nausea and vomiting

In most cases, the pain may occur when the pain is shifted downwards. It means the stone is now closer to the bladder, and that can be hazardous for your kidneys. You need to treat this health condition; otherwise, it may lead to other health-related disorders that can prove lethal for your body. If larger kidneys don’t get any appropriate treatment timely, it may lead to the other kidney problems.

How to diagnose the kidney stone?

Usually, the diagnosis of this health condition depends upon the signs that occur during the kidney stone. Your doctor may ask you about the symptoms. He or she may ask you about your urine color, family history of kidneys stone, whether you have had gout, and whether having pain in the flank. Your doctor may check the presence of red blood cells in your urine and order computed tomography (CT) scan or ultrasound of the whole urinary tract to check the presence of the kidney stone.

Treatment for the renal stone of 14mm size

Normal and smaller stone passes from the kidneys, and finally through the urethra on their own with some home remedies. In allopathy, the treatment for kidney stones usually depends upon the location, size, and damage done to the kidneys. According to the Allopathic doctors, stones larger than 6 mm require surgery. A larger kidney stone can affect the urinary tube (Ureter) and cause a problem in the urinary tract. If you have 14mm kidney stones, then allopathic doctors may recommend an urgent ureteroscopy and Holmium laser to remove the stones because it can responsible for the swelling of the kidney or infection.

You can choose Ayurvedic therapy for removing the kidney stone and avoid the re-occurrence of the disorder. Ayurvedic treatment usually works to break down the pieces of the kidney stones into smaller pieces and send them out of the body. If not managed timely, it may lead to other kidney problems. Kidney stones are also associated with the papillary renal cell carcinoma and upper tract urothelial carcinoma. They are the type of cancers that form inside the lining of the kidney tubules. Ayurvedic medicine treatment for kidney cancer is one of the best ways to manage all the kidney-related problems, especially, kidney cancer with the help of the natural herbs and some minor but significant customization in your diet.

You can reach to the Kidney and Ayurveda, where you can get the specific treatment for kidney stone with the herbs and proper diet chart that involves the proper dietary customization.

Ref: - https://tinyurl.com/rz38s4m

[kidneyayurveda]

Is it necessary to do surgery having 5.86mm kidney stones?

Introduction

Kidney stones are the deposits hard in texture and are made up of salts and minerals present inside the kidneys. Kidney stones are also known as renal lithiasis, nephrolithiasis. There are many factors responsible for the formation of kidney stones that can affect distinctive parts of the urinary tract.

Generally, the concentrated urine is the reason behind kidney stones as it allows the minerals to crystallize and get affixed together. Passing kidney stones can cause pain. However, this pain can be prevented with the help of kidney stone treatment Ayurvedic medicine.

Drinking a lot of water is the first and foremost tip to pass the stone. According to the treatment in allopathy, there are instances (stones wedged in the urinary tract) when the need for surgery arises but Ayurveda firmly disagrees with this fact. In this blog, we will read how Ayurveda can help you in the prevention of kidney stones surgery.

Population at risk

The chances of developing kidney stones in people are very common. One in every 20 to 30 people can get kidney stones. However, there are certain conditions and diseases that can lead to the development of kidney stones. People taking medications are likely to get kidney stones more than those who are living a normal life. The most common type of stone despite the gender is urinary tract stones. A kidney stone is generally non-life-threatening in nature and can come multiple times.

People living in developing countries like India commonly get stones in their bladder whereas the population in the industrialized countries get kidney stones in most common. The dietary factor is a great reason behind this cause. People suffering from hyperuricemia or high level of uric acid in the blood develop uric acid kidney stones.

Talking about the risk of kidney stones in pregnant women, it has been noticed that changes that occur during pregnancy are the major reasons behind kidney stones in such women. These changes comprise of low urine passage due to an increase in progesterone levels, low fluid consumption due to low bladder capacity. However, all these people at risk can be safeguarded with the help of kidney stone treatment Ayurvedic medicine in the USA.

Diagnosis of kidney stones

When the kidney stones began to generate symptoms then it can be diagnosed with the help of various methods. Although, there is no ideal method for the diagnosis. Imaging tests help in the confirmation of the diagnosis. CT scan, ultrasound, abdominal X-rays are some effective ways of diagnosing kidney stones. After all this examination the performance of kidney stone treatment Ayurvedic medicine becomes world-class.

Treatment of Kidney Stones

Generally, a kidney stone can be passed out of the body through the urinary tract in two days. It is possible with a huge amount of intake of water or fluid. In the western treatment method, many pain control drugs and narcotics are taken into consideration to prevent the severe pain of stones. Depending upon the sizes of the stones surgeries are suggested to the patients. But, according to Ayurveda, the kidney stone treatment Ayurvedic medicines are enough for any size of the stones and require no surgery.

The ability of body to pass a stone is affected by many factors such as the size of the stone, body size of the person, enlarged prostate, pregnancy, etc. Allopathy suggests that there are 80% chances that a stone sized 4 mm can be passed and the chances began to fall with the increasing size of the stone.

Ayurveda and kidney stones

No matter what the size of the kidney stone is, with Ayurveda, there is no need of surgeries to make these stones go out of the body. The natural preventive measures help in the reduction and removal of the kidney stones depending upon the shape, location, size, and toughness of the stone. The Ayurvedic or natural measures break the stone into smaller grains and make it easy to pass through the urine. Kidney stone treatment Ayurvedic medicines are inclusive of many such measures. Some of these are:

Eating on time.

Drinking enough water and avoiding the consumption of liquor.

Do not hold the urine for long and pee on time instead.

Drink warm water early morning. This helps in bowel cleansing.

Practice yoga and meditation.

Consume fruits with higher water content.

Ref:- https://tinyurl.com/yx8xqyuc