#multidisplinary

[studnickastudio]

[235uranium]

god that "no one respects bio" post is so... bad. biology is one of the largest fields with massive amounts of funding. bio programs are insanely competitive. what world are you living in

[healing92]

Advanced Cancer Treatment Center in JP Nagar

Supra Hospital

Being a health visitor I am always in search of finding good health care facilities for patients and their families, I got a chance to visit Supra Hospital, which is said to be the Advanced Cancer Treatment Center in JP Nagar. I came away from there with an incredible impression of their quality care, modern infrastructure, and very positive ambiance.

First Impression

From the second I walked in through the gates of Supra Hospital, I sensed this is not an ordinary hospital. Clean and modern, I would say the ambiance had such a pleasant disposition. A much-needed attribute of this Advanced Cancer Treatment Center in JP Nagar is the location being strategically placed in a way that any patient coming from any part of Bangalore will be able to reach there easily.

State-of-the-Art Oncology Department

The soul of Supra Hospital lies in its oncology department. Being the most recommended Advanced Cancer Treatment Center in JP Nagar, it is equipped with the latest diagnostic technology, precision radiation therapy, and customized chemotherapy protocols. During my visit, I felt very strongly about the fact that greater emphasis is laid on making treatment plans for cancer patients, which are highly individualized and based on the most current treatment modalities from around the world.

Multidisciplinary Team Approach

An added aspect that stands out about this Advanced Cancer Treatment Center in JP Nagar is its multidisplinary team. Involving the oncologist, radiologist, surgeon, and palliative care experts, complete treatment is handed over jointly. I have been privileged to witness collaborative discussions during my rounds among professionals as they discussed complicated cases to ensure the best course of action for their patients.

Patient-Centric Approach

What really makes Supra Hospital the leading Advanced Cancer Treatment Center in JP Nagar is their approach to big-picture care. They provided patients and their families with support from psycho-social counseling to nutritional guidance. I talked to some patients and caregivers, and they all satisfied with the staff’s empathy and responsiveness.

Technological Advancement and Facilities

The advanced means of Supra Hospital are based on the state-of-the-art technology to fight cancer: PET-CT scan, robotic surgery, immunotherapy. It is, therefore, unquestionable that many specialists would refer their patients here, known as the best Advanced Cancer Treatment Center in JP Nagar.

Community Outreach and Education

Another service that Supra Hospital proudly offers in addition to treatment is public awareness of cancer prevention and early detection. Their programs for the community and health camps testify to their concern for public health-accrediting them as a reliable Advanced Cancer Treatment Center in JP Nagar.

Final Thoughts

I spent an entire day at Supra Hospital and can say without any hesitation that it is truly the premier Advanced Cancer Treatment Center in JP Nagar. If you are looking for treatment for yourself or for a loved one, it is only Supra hospital with its unmatched expertise and a truly heartfelt approach to care that can be considered a first choice in cancer care in the region.

[nursingwriter]

Marfan syndromme is a multisystem disease with variable genotypic and phenotypic appearance. There is overlap in clinical presentation with other multisystem diseases that often may lead to misdiagnosis. Consequently because of its diverse clinical features diagnosis is challenging and necessitates a multidisplinary approach. Genetic counselling is costly, involves a lot of time consumption and does not offer a solution for the diagnostics predication. Therefore failure to make a diagnosis has social as well as medical repercussions for the individual as well as his family. Marfan syndrome is a genetic disorder of connective tissue that is inherited as an autosomal dominant disease. The spectrum of this multisystem disease mainly but not exclusively affects the musculoskeletal, the ocular and the cardiovascular systems. The disease is a fibrinllinopathy that results from defective synthesis of fibrillin 1. The gene coding for this protein FBN1 was localized to chromosome 15q21 among the human species. However another gene MSF2 located at chromosome 3p25 has been implicated. Marfan affects 1 in 1000 (Pyeritz et al. 1981). In the United States of America it is estimated that at least 200000 people suffer from it or related connective tissues disorders. This makes it one of the most common single gene malformations. The is no geographical distributions and it affects people of all races. The earliest recordable history of this disease dates back to 1986 when a Paediatric Professor Antoine Marfan, presented the case of a 5-year-old girl to the Societe Medicale des Hapitaux de Paris (Marfan; 1896). The child Gabrielle P. had conspicuous abnormalities of her skeletal system that developed till the time of her demise probably from tuberculosis. (Marfan, 1938) It cannot be proved whether or not Gabrielle suffered from Marfan syndrome. Veritably she may have been suffering from congenital contractual arachnodactly. (Hecht and Beals, 1972). During the 20th century further phenotypical manifestations of Marfan's syndromme were discovered. For example ectopia lentis (Borger, 1914), the fact that inheritance is autosomal dominant (Weve, 1931). Aortic dissection as part of the cardiovascular involvement (Etter and Glover1943). Dilatation (Baer et a1., 1943). Involvement of mitral valve prolapse (Brown et al., 1975; ) and Ectasia of the dura (Pyeritz et al., 1988). The gene associated with Marfan syndromme was identified by Francesco Ramirez in 1991 at the Mount Sinai Medical centre in New York. Until recently the diagnosis of Marfan's syndrome relied on a clinical criteria developed in 1986 in Berlin. It is commonly known as the Berlin Nosology or Berlin Criteria (Beighton et al., 1988). Because the Berlin Criteria did not provide for molecular data and led to misdiagnosis of relatives who were unaffected a new criteria, the Ghent criteria was devised in 1995. Diagnosis of Marfan syndrome is in evolution. Existing criteria are in constant revision based on a workshop that took place in 2007 in Brussels Belgium. This paper is going to discuss the diagnostic challenge of Marfan syndromme. PRESENTATION Marfan syndromme is a multisystem disease that involves the skeletal, cardiovascular, ocular, pulmonary and the skin and integumentary system. The skeletal system involves a disproportion of the limbs and digits. A condition scientifically known as dolichostenomelia. This is more marked by the concomitant underdevelopment of muscle and fat. Sufferers of this syndromme characteristically appear very tall and thin with broad arms. Abnormalities of the limb length are measured as a ratio of the upper segment to the lower segment. The lower segment involves measurement from the floor to the top of the symphsis pubis while the upper segment is obtained by subtracting the lower segment from the total height. In the normal adult population this ratio is 0.93. However is a patient with Marfan's it is 0.85. Arachnodactly or spider web fingers is another salient feature of skeletal abnormalities linked to marfan's. Clinically this is assessed by the Walker- Murdoch Thumb sign. The index finger and the thumb encircle the contra lateral wrist with overlap of the distal phalanges. Deformities of the anterior chest commonly known as pes excavatum or a funnel shaped chest. The converse pes carinatum or protrusion of the chest may also appear as a presentation. Hyper mobility of joints is a common trait as well. Often it is exaggerated in the arms and presents as flat foot in the feet. Spinal scoliosis of greater than 20 degrees or sideways curvature of the vertebral column is another skeletal manifestation. ( Sponseller et al. 1995) it is often complex and is characterised with the disappearance of the dorsal kyphosis leading to a flat back. In addition there are asymptomatic basilar impressions, atlantoaxial subluxation and lumbar spondilolysthesis.( Jean Marie Le Parc, 2003) craniofacial abnormalities are another manifestation. Malar hypoplasia, anterior posterior axis dolicocephalia and micrognathia. On clinical assessment of the mouth an arched palate is often found, crowding of the teeth and extreme maxillary ovejet ( overbite). About 70% of patients require treatment at an orthodontist. (Westling et al., 1998) Protrusio acetabuli or protrusion of the acetabular is also present . It is a malpresentation of the hip joint where the medial wall of the acetabulum invades the pelvic cavity with associated medial displacement of the femoral head. And This is assessed using radiography. It affects 31-100% of patients to varying degrees ( Sponseller et al., 1995) Clinical presentation include hip joint stiffness and gradual limitation in activity related to joint pain, a waddling gait, restricted range of motion, flexion contracture, a pelvic tilt leading to hyperlordosis of the lumbar spine, and finally osteoarthritic changes. The consequence is early hip pain and osteoarthritis Neuromeningeal involvement in form of dural ectasia or dilation of the dural sac is not a skeletal manifestation in strict terms. However its ramifications primarily affect the spine often leading to lumbar scalloping. 92% of patients were found to have dural ectasia (Fattori et al. 1999) Magnetic resonance imaging and CT scan was found to have high sensitivity and specificity in its diagnosis ( Ahn et al. 2000). Cardiovascular system involvement is the most serious complication related to Marfan's syndromme. It presents with dilation of the aortic root and involves dilatation of the sinuses of valsalva. Its prevalence is about 70-80%. The major life threatening complications are aortic root dilatation and aortic dissection. Baer et al. (1943) and Etter and Glover (1943). A third of the patients were found to have prolapse of the mitral valve mitral enlargement of the aortic root or a combination of both on echocardiography this was despite normal auscultatory finding on clinical examination. ( Brown et al., 1975) Manifestation is at an early age and is more common in men than it is in women. Other features include dilation of the main pulmonary artery and the descending abdominal aorta. Calcification of the mitral annulus. The prevalence of ventricular and supraventricular arrhythmias also seems to be higher in patients with Marfan's syndromme than in the general population. With regard to ocular manifestation the major presentation is ectopia lentis. This is a subluxation or malposition of the lens of the eyes. Commonly known as subluxation the lens is displaced in a supero- temporal direction. Presentation may be at Birth or may develop during childhood or adolescence. Other manifestations include a flat cornea. An increase in the axial length of the globe. Formation of cataracts or opacification of the lens. Detachment of the retina, near sightedness and Glaucoma or increase in intraocular pressures. Involvement of the skin and integumentary system is manifested as striae atrophicae or stretch marks in the absence of weight gain. Manifestations of the pulmonary system include spontaneous pneumonthorax or air in the chest cavity and multiple pneumonic blebs found on chest radiograph (Yellin et al., 1991). CLINICAL DIAGNOSIS The clinical diagnosis of Marfan syndromme has progressive changed throughout the years. Both clinical criteria and molecular criteria are used in the correct diagnosis of Marfan syndromme. The first tool used for diagnosis was the Berlin Nosology or Criteria. (Beighton et al. 1988.) This criteria was laid out during the 7th International Conference on Human Genetics workshop that was held in Berlin. It was composed of Major criteria. These were composed of features that were not commonly found in the general population and therefore carried a lot of weight in diagnosis. Minor criteria involved features that were common in the general population and that could also be confused with conditions that mimicked Marfan. The Berlin Nosology stated that in the absence of an unequivocally affected first-degree relative, one should require involvement of the skeleton and at least 2 other systems with a minimum of 1 major manifestation (ectopia lentis, aortic dilatation or dural ectasia). In the presence of an unequivocally affected first-degree relative, one requires only that 2 organ systems be involved. The Berlin Nosologogy had 6 systems. The skeletal, the cardiovascular, the ocular, the pulmonary, the Skin and Integumentary and the central nervous systems . There were only 2 major criteria Ectopia of the Lens in the ocular system and aortic dilation or aortic dissection in the cardiovascular system. Other criteria were classified as minor criteria across the six systems. As time went by weaknesses in the Berlin Nosology became evident. This became more so with the advancement of molecular Biology Tests. Subsequently a group of clinicians met in Ghent Belgium and came up with the current diagnostic criteria known as the Ghent Nosology. (De Paepe et al. 1996) Similar to the Berlin Nosology the Ghent criteria was based on clinical findings in the various organ systems as well as the nature of family history and relationships, a major criteria was classified as which has a high diagnostic specificity because it was less frequent in other conditions and in the general population. A point of divergence from the Berlin Nosology was the conversion of minor criteria in the skeletal system into major criteria. For one to be diagnosed with Marfan's the patient must have a first degree relative diagnosed with the disease in addition two systems must be involved with one having a major sign. In the absence of a family history or genetic criteria three systems have to be involved two with major signs. In addition with the Ghent nosology there was a major demarcation between a major criterion being present and the system being involved (De Paepe et al. 1996). The Ghent Nosology was divided into seven systems. Skeletal Ocular, Cardiovascular, Pulmonary, Cutaneous, Dura Mater, and Genetic. Major clinical signs in this criteria with regard to the skeletal system include Pes carinatum or Pes excavatum requiring surgery, Upper segment to lower segment or an arm span ratio of greater than 0.5. wrist or thumb sign. Scoliosis of > than 20 degrees or spondylolisthesis. Acetabulum protrusion. Flat feet and elbow extension of less than 170 degrees. Contrary to the previous system lumbosacral ectasia has been included as a major criteria in the Dura Mater system. A direct parent meeting the diagnostic criteria, Mutation of FBN1 gene known to cause Marfan or the presence of a genetic marker that is close to FBN1 and is transmitted with the disease in the family is also regarded as major criteria. As previously the presence of a major criteria does not necessarily mean system involvement. More stringent measures were put in place. With regard to the skeletal system a system is involved if there is presence of at least four major clinical signs. Involvement in the ocular system means presence of at least 2 minor signs. The cardiovascular, the pulmonary, and the cutaneous are involved if the is presence of at least one minor sign respectively. While the dura mater is involved if there is presence of at least one major sign. Treatment. In General all patients with Marfan's syndromme should have their physical activity restricted, they should also be put on prophylaxis for endocarditis. In additional they should have an annual echocardiography check up and be put on treatment with Beta Blocking agents. The mainstay of therapy in management of the cardiovascular system is the use of Beta Blockers. This was discovered in the early 1970's where the use of Beta Blockers was found to reduce the incidence of developing aortic dissection. In addition Beta blockers retard aortic growth in both children and adults (McKusick, 1972) and ( Shores et al., 1994). In children an annual echocardiography is recommended. When the diameter of the aortic root is greater than 5 cm prophylactic surgery is highly recommended. Furthermore the success rate of Surgery has greatly improved with very minimal mortality rates reported. (McDonald et al. 1981) ACE inhibitors or Angiotensin converting enzyme inhibitors have also been found to improve the condition of patients with Marfan's syndromme. They have been found to reduce central arterial pressures and aortic stiffness. Progesterone and Estrogen therapy has also been found to reduce the patient's height if therapy is begun before puberty. Patients with Marfan's may also require Orthopaedic surgery, ophthalmic surgery and treatment Pneumothorax surgery as well as genetic counselling. Finally these patients may also need psychiatric evaluation. DISCUSSION Diagnosis of Marfan syndromme is to say the least challenging. A multidisplinary approach is required. An orthopaedic specialist, a cardiologist, an ophthalmologist a geneticist, a radiographer and a specialist nurse are part of the team involved in the diagnosis of Marfan. Engaging this myriad of specialists can be costly to the sufferers of Marfan syndromme. To diagnose Marfan the entire FBN1 gene has to be screened. This process is expensive and is only available privately even in developed countries. Furthermore genetic testing has a success rate of 70-80% and cannot wholly exclude marfan's. Skeletal features of Marfan's syndromme can be difficult to define.. The graphs define upper and lower body ration are age dependent and are not commonly available. Moreover they only provide mean values without standard deviation making their interpretation challenging. The usefulness of span greater than height has been put into question in the diagnosis of marfan's syndromme. (Schott, 1992) the concept which was a trademark with Leonardo da Vinci allows man to be assessed or portrayed in a square. However anthropometric studies have revealed that span exceeds height by a value of 59-78% in the normal adult population. In addition assessing whether pes carinatum or pes excavatum is mild moderate or severe is difficult clinically. Similarly features that commonly lead to referral like a tall thin body are not included in the diagnostic criteria neither can they be used to discriminate against patients with marfan's syndromme. It is also important to note that the progress and appearance of symptoms of Marfan's over time makes the diagnosis difficult. All features do not appear at once and evolve over time from child hood to adolescence. There are conditions have clinically presented themselves like marfan's syndromme and have clinical diagnosis a difficulty. For Instance the MASS syndromme. (Glebsy and Pyeritz 1989) This disease presents with Mitral Valve Prolapse. This is a situation where the mitral valve of the heart closes properly but allows blood to regurgitate into the heart during ventricular contraction. The diameter of the Aortic root may be towards the upper limit of normal however there is no progression to an aneurysm neither is there development of aortic dissection. There are also Stretch marks of the skin that are unrelated to weight gain. Finally the disease presents with skeletal features of marfan's like scoliosis joint hyper mobility. MASS syndromme is a connective tissue disorder. It is inherited in an autosommal dominant pattern and is associated with mutations of Fibrillin 1 gene. FBN1. Family history may be positive however patients with MASS syndromme never develop dislocation of the lens or aortic root enlargement. Another condition is Stickler syndromme or Hereditary athroopthalmopathy (Stickler et al. 1965) This disorder is also multisystem. Its diagnosis requires the eye. Craniofacial and another system should be affected. Salient features of this disease include myopia degeneration of the retina and the vitreous, detachment of the retina, deafness athropathies, hyper mobility of the joints facial hypoplasia and microganathia. Most of these features are shared with Marfan's syndromme . In Shprintzen -- Goldberg syndromme (Shprintzen and Goldberg 1982) there are skeletal changes that are suggestive of Marfan syndromme. However these patients have craniosynostosis and neurodevelopmental abnormalities. In addition aortic dilatation may be present. Other conditions are Congenital Contractual Arachdinodactyly (Viljoen 1994) .Familial Aortic Dissection,( Nicod et al. 1989) Familial Ectopic lentis (Tsipouras et al. 1992) and Familial Marfan like Habitus ( Milewicz et al. 1995) In some countries or some clinical setting either the Ghent criteria or the Berlin Nosology is still employed in diagnosis. Prior to advancement of molecular biology diagnosis of Marfan's depended on the Berlin Nosology. However this criteria was not strict in its approach and at times lead to over diagnosis of patients with Marfan's syndromme. (Pereira et al. 1994) This was further emphasised by ( Rose et al. 2000) in this study 19% of patients who were diagnosed under the Berlin Nosology failed to pass the test of the Ghent nosology. In addition this study came up with an interesting finding. Dural ectasia was found to be the second most common major diagnostic manifestation. It was found out that screening for dural ectasia established the diagnosis of Marfan of syndromme in 23% of patients under the Ghent Nosology. Another glaring weakness of the Berlin Nosology was that it encouraged the diagnosis of Marfan using non-specific criteria once the diagnosis of Marfan was made in a first degree relative. In this scenario the only requirement for a diagnosis to be made was the identification of a trait commonly seen in Marfan patients in two organ systems. Problems of over diagnosis and Misdiagnosis arose. The present proposal is more stringent in that, in addition to a family history of Marfan syndrome (which still means identification of an individual in the family who independently satisfies diagnostic criteria on clinical grounds alone), the presence of a major clinical manifestation and involvement of a second system are required for definitive diagnosis. The Ghent criteria aims to be as objective as possible in selecting those with Marfan's syndromme. However several shortcomings have been noted. For instance most manifestations are age related and some as earlier discussed are difficult to quantify. In addition few studies have come up to test the sensitivity and specificity of Marfan's syndromme anomalies. For example only recently has the association between dural ectasia and Marfan syndromme been pointed out. (Pyeritz et al. 1981) Little is Known about the Prevalence of dural ectasia and further studies are therefore needed to qualify this presentation as a major criteria. There are also psychological issues that one has to undergo through when they are labelled to Have Marfan syndromme. Read the full article

[hunkydorkling]

Part 1 of my trip to the mountains of Baguio in photos and possibly one video, in no particular order:

Vegan rooftop restaurant! These dishes taste exquisitely like the real thing, and are mostly made of tofu, eggplant, and sobafien.

The place we went to also housed decorative art that's part of multidisplinary artist Kidlat Tahimik's exhibit called Now More Than Ever. I managed to shoot photos of my favorite piece instead because it was all I could afford lol

Hello, it's your queerpal Corny. Howdydo.

And below you will see an assortment of artworks from the Bencab Museum:

[researchupdates]

Global Sleep Bruxism Market May See a Big Move by 2028

Overview of Global Sleep Bruxism Market:

Today’s businesses choose the market research report solution such as Sleep Bruxism Market report because it lends a hand with the improved decision making and more revenue generation. The report also aids in prioritizing Market goals and attain profitable business. Industry tendencies, the growth proportion of major producers, and production analysis are the segments included in the chapter of global growth trends of Sleep Bruxism business report. While studying Market size by application it covers Market consumption analysis by application whereas studying Market size by type includes analysis of value, product utility, Market percentage, and production Market share by type.

The persuasive Sleep Bruxism Market report is all-embracing of the data which covers Market definition, classifications, applications, engagements, Market drivers, and Market restraints that are based on the SWOT analysis. Analysis of profiles of manufacturers or commanding players of the global Market is performed based on sales area, key products, gross margin, revenue, price, and production. The chapter of Market value chain and sales channel analysis of this Market document includes details of customer, distributor, Market value chain, and sales channel analysis. An excellent Sleep Bruxism report consists of most recent Market information with which companies can attain in depth analysis of Healthcare industry and future trends.

Available Exclusive Sample Copy of this Report @ https://www.databridgemarketresearch.com/request-a-sample/?dbmr=global-sleep-bruxism-market .

The Global Sleep Bruxism Market is expected to growing at a healthy CAGR of 6.10% in the forecast of 2021 to 2028.

According to the market report analysis, Sleep Bruxism is defined as a repetitive clenching and grinding activity of jaw muscles, characterized by oral parafunction at subconscious level, its prevalence is found to be equally distributed amongst males and females as reported by Peter Wetselaar in his study over Dutch population. Awareness regarding benefits of early detection of sleep bruxism would be helpful in avoiding damage to oral and face structure as well as for dental diagnostics market associated with sleep bruxism market.

Some of most important key factors driving the growth of the Global Sleep Bruxism Market are emerging market and huge investments in research and development, sleep Bruxism treatment demands multidisplinary approach and increasing awareness about oral health, high healthcare expenditure across the globe.

The key players in the Sleep Bruxism Marketare Pfizer Inc., Randmark Dental products, Akervall Technologies, Patterson Dental Supply, Henry Schein, Carestream Dental, S4S Dental Laboratory, Merz Pharma GmbH & Co., PLANMECA OY, Ipsen Biopharmaceuticals, Dental Lab India, Bupa, American Dental Craft, Polpharma, Covis Pharma, Norwich Pharma Services, Teva Pharmaceutical Industries Ltd., Oventus, Energy Resources International Co., Ltd., Implants Broadcast International, Bio 3 Implants GMBH, BioMedical Industry Group Inc., among other domestic and global players.

Access Complete Report @ https://www.databridgemarketresearch.com/reports/global-sleep-bruxism-market .

[evoldir]

Fwd: Postdoc: Valencia.SARS-CoV-2_Evolutionary

Begin forwarded message: > From: evoldir@evol.biology.mcmaster.ca > Subject: Postdoc: Valencia.SARS-CoV-2_Evolutionary > Date: 7 May 2021 at 06:51:28 BST > To: rdmpage@gmail.com > > > > BIOINFORMATICS POSTDOC IN SARS-CoV-2 EVOLUTIONARY GENOMICS > > Institute for Integrative Systems Biology (I2SysBio), UV-CSIC, Valencia, > Spain > > A postdoctoral position (1.5 years) is available immediately in > the PathoGenOmics group of Mireia Coscolla in Valencia (Spain) > www.uv.es/pathogenomic > > We are seeking enthusiastic applicants with skills in computational > biology/bioinformatics and with experience in data mining and comparative > or evolutionary genome analyses. Skills in Bayesian phylogenetics and > phylodynamics, and pathogen evolution is a plus. > > The project: Strategic initiative for genomic surveillance and assessment > of the impact of SARS-CoV-2 mutations in real time. The aim of the > position is to investigate molecular epidemiology dynamics, mutations > surveillance, and to collaborate with a multidisplinary team to explore > the functional impact of the different SARS-CoV2 variants. The project > is highly linked to the ongoing project https://ift.tt/34AyKAj. > > The tasks of the postdoc will be in sequence and evolutionary analyses > of SARS-CoV-2 sequences within a consortium with eight research groups > ranging from virology to mathematics and protein structure.  The position > is based in Valencia, at the Institute for Integrative Systems Biology > (I2SysBio), a mixed institute between university of Valencia and > CSIC. The Pathogenomics group is part of the Pathogen Systems Biology > program, and we study microbial pathogens in the context of their host and > disease. To approach this, we use a range of different omics technologies > where genomics is our main approach to study the evolution and molecular > epidemiology of the pathogen. But we also perform experimental infections > to discover which genomic determinants are involved in different virulence > readouts, using among other approaches transcriptomic of host and pathogen > To apply send CV and e-mail of two references to Mireia.coscolla@csic.es > > > Mireia Coscollá Devís: > www.mireiacoscolla.com > Principal investigator: > www.uv.es/pathogenomic > I2SYSBIO, Parc Cientific - Universitat de València > C/Agustín Escardino, 9, 46980 Paterna (Valencia) > Telephone.: 0034 963543317 > e-mail: > mireia.coscolla@uv.es > > > > Mireia > via IFTTT

[adalidda]

Illustration Photo: a couple of fisherman harvested pink algae for use in cosmetic (Image by Quang NGUYEN DANG from Pixabay)

Blue Entrepreneurship Programme 2021 for Ireland, France & Spain

In the Framework of the MarENet project, we are launching a Blue Entrepreneurship Programme to foster innovative business ideas in the Blue Economy sector across the Atlantic Frontage (Ireland, France & Spain).

Through the implementation of this entrepreneurship programme, the MarENet consortium seeks to strengthen candidate’s business skills and help them to develop the tools required for implementing a new business idea related to the marine environment.

This program of mentoring and training for entrepreneurship will have a duration of approximately 10 months, and will provide participants with the tools and skills required to develop their business ideas into potential realities.

As a participant you will have the opportunity to

Acquired business skills

Participating in the different training courses conducted by experts on how to prepare a business model, a financial plan, pitch to investors, marketing and branding plan..

Gain technical knowledge

Experts from our multidisplinary consortium will conduct webinars on different topics in the most representative Blue Economy sectors like shipbuilding, fisheries, ports, aquaculture, biotechnology...

Participate on a blue hackathon

After a competition phase, the selected projects will have the opportunity to participate in a European Blue Hackathon in Cork, Ireland. This event will give you the opportunity to interact with key stakeholders from the sea-port environment.

Potential funding opportunities

The selected projects after the Blue Hackathon competition will receive a close assitance to identify potential funding opportunities and synergies with the industry to put into practice their ideas.

Application Deadline: February 28th, 2021

Check more https://adalidda.com/posts/BypHB5Cu8vEqK4x53/blue-entrepreneurship-programme-2021-for-ireland-france-and

[ownerzero]

New multidisplinary exhibition set in an estate agents explores the housing crisis

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The housing market, as we all know, is sort of, slightly, maybe, a little bit…well, screwed. In major cities, at least. Amanda Lwin, curator of Unreal Estates, a group show which opens in an estate agent’s office in east London…

The post New multidisplinary exhibition set in an estate agents explores the housing crisis appeared first on AWorkstation.com.

source https://aworkstation.com/new-multidisplinary-exhibition-set-in-an-estate-agents-explores-the-housing-crisis/

[4rtmorelikef4rt]

David Antonio Cruz

David Antonio Cruz is a multidisplinary artist that focuses on the beauty and representation of black, brown and queer bodies in his work. Through his paintings and performances, he reflects on themes of sexuality, race, and representation. Cruz’s’ Puerto Rican culture and sexual preference influence his want to “queerify” the subjects in his pieces which refers to the practice of accepting images and altering them.

Cruz works closely with color in his paintings. Specifically by thinking about how the colors temperature affect the viewers as they interact with the work. In his piece “SOLETTHEMEATASYLUMPINK”, he paints the two panels of the background different shades of pink that both bring in and interact with the viewer as they see the painting from various angles. The “wegivesomuchandgivenothingatall” series of paintings had the most impact on me. In this series, he memorializes trans women who were murdered in between 2017 and 2018. The reappropriation of their images into renditions of fashion magazine covers was a way to remember these women by their beuaty and not by their deaths. His use of color and absence of color allows the viewer to pay attention to the women’s faces, and the rich beautiful skin tones that Cruz was able to recreate through this medium.

Another important aspect of Cruz’s, practice is performance pieces. A variety of topics are elabortaed upon in these performances but they all have the common idea of ‘The Duende’. As explored in the “Theory and Play Of The Duende”, the idea of duende is present in indigenous, greek and Spanish cultures that aims to explore the idea of an unexplainable surge of emotion; “The duende is not in the throat: the duende surges up, inside, from the soles of the feet.’ Meaning, it’s not a question of skill, but of a style that’s truly alive: meaning, it’s in the veins: meaning, it’s of the most ancient culture of immediate Creation”. Cruz states that when he performs his pieces, for example, “howtoorderachocolatecake”, he finds the duende within himself when he lets himself get consumed by his inner emotions.

Cruz’s work is a culmination of his identities and experiences. Much like the point that Taiye Selasi”s was making in her TedTalk “Don’t ask where I’m from, ask where I’m a local”, we are from where we form our experiences. Cruz incorporates aspects of his Puerto Rican upbringing such as preforming pieces in Spanish or incorporating Purto Rican phrases in his pieces, with his role in the gay community that influences the presentation of the subjects he chooses to paint and the topics that are discussed in his performance pieces.

[prasanthvarma]

Gastroenterology - Regency Healthcare

Delivering patient-centred digestive healthcareRegency Healthcare is dedicated to prevention, diagnosis, treatment & management of all types of digestive diseases. Possessing cutting-edge expertise and latest diagnostic & endoscopic technology and by offering patient-centric treatment plans through a team of multidisplinary specialists; our Gastroentrology department provides the best personalised care for a wide range of gastrointestinal conditions including stomach ulcers, gallstones and irritable bowel syndrome among others.

Types of Gastrointestinal diseases

Causes

Symptoms

Gastrointestinal bleeding

Stomach cancer

Irritable bowel syndrome (IBS)

Gastroesophageal reflux disease (GERD)

Peptic Ulcers

Also known as gastrointestinal hemorrhage, GI bleed majorly occurs from the mouth to the rectum or gastrointestinal tract.

It is the fifth-most common cancer worldwide that can include colorectal cancer, gastric cancer, pancreatic cancer, liver cancer and more.

IBS is a chronic condition that is quite common in nature and affects the large intestine. Bloating, constipation are few common symptoms of the same.

It refers to a digestive disorder that affects the lower esophageal sphincter (LES) causing the stomach’s contents to flow back into the esophagus. Though in most cases, it can be relieved through diet and lifestyle changes, but in some cases a patient may require to take help of medication or surgery.

Peptic ulcers are kind of sore that form in the lining of the stomach or small intestine as a result of inflammation caused by excessive stomach acid. A common health problem, it can be relieved with the help of medication & proper care.

Our Top Ranked Team Of ExpertsThe core strength of Regency Healthcare is our team of highly trained super specialist doctors from prestigious institutes with expertise in managing a wide spectrum of gastro ailments. They not only believe in doing the best for you, but also achieving the best for everyone.

[nursingwriter]

Marfan syndromme is a multisystem disease with variable genotypic and phenotypic appearance. There is overlap in clinical presentation with other multisystem diseases that often may lead to misdiagnosis. Consequently because of its diverse clinical features diagnosis is challenging and necessitates a multidisplinary approach. Genetic counselling is costly, involves a lot of time consumption and does not offer a solution for the diagnostics predication. Therefore failure to make a diagnosis has social as well as medical repercussions for the individual as well as his family. Marfan syndrome is a genetic disorder of connective tissue that is inherited as an autosomal dominant disease. The spectrum of this multisystem disease mainly but not exclusively affects the musculoskeletal, the ocular and the cardiovascular systems. The disease is a fibrinllinopathy that results from defective synthesis of fibrillin 1. The gene coding for this protein FBN1 was localized to chromosome 15q21 among the human species. However another gene MSF2 located at chromosome 3p25 has been implicated. Marfan affects 1 in 1000 (Pyeritz et al. 1981). In the United States of America it is estimated that at least 200000 people suffer from it or related connective tissues disorders. This makes it one of the most common single gene malformations. The is no geographical distributions and it affects people of all races. The earliest recordable history of this disease dates back to 1986 when a Paediatric Professor Antoine Marfan, presented the case of a 5-year-old girl to the Societe Medicale des Hapitaux de Paris (Marfan; 1896). The child Gabrielle P. had conspicuous abnormalities of her skeletal system that developed till the time of her demise probably from tuberculosis. (Marfan, 1938) It cannot be proved whether or not Gabrielle suffered from Marfan syndrome. Veritably she may have been suffering from congenital contractual arachnodactly. (Hecht and Beals, 1972). During the 20th century further phenotypical manifestations of Marfan's syndromme were discovered. For example ectopia lentis (Borger, 1914), the fact that inheritance is autosomal dominant (Weve, 1931). Aortic dissection as part of the cardiovascular involvement (Etter and Glover1943). Dilatation (Baer et a1., 1943). Involvement of mitral valve prolapse (Brown et al., 1975; ) and Ectasia of the dura (Pyeritz et al., 1988). The gene associated with Marfan syndromme was identified by Francesco Ramirez in 1991 at the Mount Sinai Medical centre in New York. Until recently the diagnosis of Marfan's syndrome relied on a clinical criteria developed in 1986 in Berlin. It is commonly known as the Berlin Nosology or Berlin Criteria (Beighton et al., 1988). Because the Berlin Criteria did not provide for molecular data and led to misdiagnosis of relatives who were unaffected a new criteria, the Ghent criteria was devised in 1995. Diagnosis of Marfan syndrome is in evolution. Existing criteria are in constant revision based on a workshop that took place in 2007 in Brussels Belgium. This paper is going to discuss the diagnostic challenge of Marfan syndromme. PRESENTATION Marfan syndromme is a multisystem disease that involves the skeletal, cardiovascular, ocular, pulmonary and the skin and integumentary system. The skeletal system involves a disproportion of the limbs and digits. A condition scientifically known as dolichostenomelia. This is more marked by the concomitant underdevelopment of muscle and fat. Sufferers of this syndromme characteristically appear very tall and thin with broad arms. Abnormalities of the limb length are measured as a ratio of the upper segment to the lower segment. The lower segment involves measurement from the floor to the top of the symphsis pubis while the upper segment is obtained by subtracting the lower segment from the total height. In the normal adult population this ratio is 0.93. However is a patient with Marfan's it is 0.85. Arachnodactly or spider web fingers is another salient feature of skeletal abnormalities linked to marfan's. Clinically this is assessed by the Walker- Murdoch Thumb sign. The index finger and the thumb encircle the contra lateral wrist with overlap of the distal phalanges. Deformities of the anterior chest commonly known as pes excavatum or a funnel shaped chest. The converse pes carinatum or protrusion of the chest may also appear as a presentation. Hyper mobility of joints is a common trait as well. Often it is exaggerated in the arms and presents as flat foot in the feet. Spinal scoliosis of greater than 20 degrees or sideways curvature of the vertebral column is another skeletal manifestation. ( Sponseller et al. 1995) it is often complex and is characterised with the disappearance of the dorsal kyphosis leading to a flat back. In addition there are asymptomatic basilar impressions, atlantoaxial subluxation and lumbar spondilolysthesis.( Jean Marie Le Parc, 2003) craniofacial abnormalities are another manifestation. Malar hypoplasia, anterior posterior axis dolicocephalia and micrognathia. On clinical assessment of the mouth an arched palate is often found, crowding of the teeth and extreme maxillary ovejet ( overbite). About 70% of patients require treatment at an orthodontist. (Westling et al., 1998) Protrusio acetabuli or protrusion of the acetabular is also present . It is a malpresentation of the hip joint where the medial wall of the acetabulum invades the pelvic cavity with associated medial displacement of the femoral head. And This is assessed using radiography. It affects 31-100% of patients to varying degrees ( Sponseller et al., 1995) Clinical presentation include hip joint stiffness and gradual limitation in activity related to joint pain, a waddling gait, restricted range of motion, flexion contracture, a pelvic tilt leading to hyperlordosis of the lumbar spine, and finally osteoarthritic changes. The consequence is early hip pain and osteoarthritis Neuromeningeal involvement in form of dural ectasia or dilation of the dural sac is not a skeletal manifestation in strict terms. However its ramifications primarily affect the spine often leading to lumbar scalloping. 92% of patients were found to have dural ectasia (Fattori et al. 1999) Magnetic resonance imaging and CT scan was found to have high sensitivity and specificity in its diagnosis ( Ahn et al. 2000). Cardiovascular system involvement is the most serious complication related to Marfan's syndromme. It presents with dilation of the aortic root and involves dilatation of the sinuses of valsalva. Its prevalence is about 70-80%. The major life threatening complications are aortic root dilatation and aortic dissection. Baer et al. (1943) and Etter and Glover (1943). A third of the patients were found to have prolapse of the mitral valve mitral enlargement of the aortic root or a combination of both on echocardiography this was despite normal auscultatory finding on clinical examination. ( Brown et al., 1975) Manifestation is at an early age and is more common in men than it is in women. Other features include dilation of the main pulmonary artery and the descending abdominal aorta. Calcification of the mitral annulus. The prevalence of ventricular and supraventricular arrhythmias also seems to be higher in patients with Marfan's syndromme than in the general population. With regard to ocular manifestation the major presentation is ectopia lentis. This is a subluxation or malposition of the lens of the eyes. Commonly known as subluxation the lens is displaced in a supero- temporal direction. Presentation may be at Birth or may develop during childhood or adolescence. Other manifestations include a flat cornea. An increase in the axial length of the globe. Formation of cataracts or opacification of the lens. Detachment of the retina, near sightedness and Glaucoma or increase in intraocular pressures. Involvement of the skin and integumentary system is manifested as striae atrophicae or stretch marks in the absence of weight gain. Manifestations of the pulmonary system include spontaneous pneumonthorax or air in the chest cavity and multiple pneumonic blebs found on chest radiograph (Yellin et al., 1991). CLINICAL DIAGNOSIS The clinical diagnosis of Marfan syndromme has progressive changed throughout the years. Both clinical criteria and molecular criteria are used in the correct diagnosis of Marfan syndromme. The first tool used for diagnosis was the Berlin Nosology or Criteria. (Beighton et al. 1988.) This criteria was laid out during the 7th International Conference on Human Genetics workshop that was held in Berlin. It was composed of Major criteria. These were composed of features that were not commonly found in the general population and therefore carried a lot of weight in diagnosis. Minor criteria involved features that were common in the general population and that could also be confused with conditions that mimicked Marfan. The Berlin Nosology stated that in the absence of an unequivocally affected first-degree relative, one should require involvement of the skeleton and at least 2 other systems with a minimum of 1 major manifestation (ectopia lentis, aortic dilatation or dural ectasia). In the presence of an unequivocally affected first-degree relative, one requires only that 2 organ systems be involved. The Berlin Nosologogy had 6 systems. The skeletal, the cardiovascular, the ocular, the pulmonary, the Skin and Integumentary and the central nervous systems . There were only 2 major criteria Ectopia of the Lens in the ocular system and aortic dilation or aortic dissection in the cardiovascular system. Other criteria were classified as minor criteria across the six systems. As time went by weaknesses in the Berlin Nosology became evident. This became more so with the advancement of molecular Biology Tests. Subsequently a group of clinicians met in Ghent Belgium and came up with the current diagnostic criteria known as the Ghent Nosology. (De Paepe et al. 1996) Similar to the Berlin Nosology the Ghent criteria was based on clinical findings in the various organ systems as well as the nature of family history and relationships, a major criteria was classified as which has a high diagnostic specificity because it was less frequent in other conditions and in the general population. A point of divergence from the Berlin Nosology was the conversion of minor criteria in the skeletal system into major criteria. For one to be diagnosed with Marfan's the patient must have a first degree relative diagnosed with the disease in addition two systems must be involved with one having a major sign. In the absence of a family history or genetic criteria three systems have to be involved two with major signs. In addition with the Ghent nosology there was a major demarcation between a major criterion being present and the system being involved (De Paepe et al. 1996). The Ghent Nosology was divided into seven systems. Skeletal Ocular, Cardiovascular, Pulmonary, Cutaneous, Dura Mater, and Genetic. Major clinical signs in this criteria with regard to the skeletal system include Pes carinatum or Pes excavatum requiring surgery, Upper segment to lower segment or an arm span ratio of greater than 0.5. wrist or thumb sign. Scoliosis of > than 20 degrees or spondylolisthesis. Acetabulum protrusion. Flat feet and elbow extension of less than 170 degrees. Contrary to the previous system lumbosacral ectasia has been included as a major criteria in the Dura Mater system. A direct parent meeting the diagnostic criteria, Mutation of FBN1 gene known to cause Marfan or the presence of a genetic marker that is close to FBN1 and is transmitted with the disease in the family is also regarded as major criteria. As previously the presence of a major criteria does not necessarily mean system involvement. More stringent measures were put in place. With regard to the skeletal system a system is involved if there is presence of at least four major clinical signs. Involvement in the ocular system means presence of at least 2 minor signs. The cardiovascular, the pulmonary, and the cutaneous are involved if the is presence of at least one minor sign respectively. While the dura mater is involved if there is presence of at least one major sign. Treatment. In General all patients with Marfan's syndromme should have their physical activity restricted, they should also be put on prophylaxis for endocarditis. In additional they should have an annual echocardiography check up and be put on treatment with Beta Blocking agents. The mainstay of therapy in management of the cardiovascular system is the use of Beta Blockers. This was discovered in the early 1970's where the use of Beta Blockers was found to reduce the incidence of developing aortic dissection. In addition Beta blockers retard aortic growth in both children and adults (McKusick, 1972) and ( Shores et al., 1994). In children an annual echocardiography is recommended. When the diameter of the aortic root is greater than 5 cm prophylactic surgery is highly recommended. Furthermore the success rate of Surgery has greatly improved with very minimal mortality rates reported. (McDonald et al. 1981) ACE inhibitors or Angiotensin converting enzyme inhibitors have also been found to improve the condition of patients with Marfan's syndromme. They have been found to reduce central arterial pressures and aortic stiffness. Progesterone and Estrogen therapy has also been found to reduce the patient's height if therapy is begun before puberty. Patients with Marfan's may also require Orthopaedic surgery, ophthalmic surgery and treatment Pneumothorax surgery as well as genetic counselling. Finally these patients may also need psychiatric evaluation. DISCUSSION Diagnosis of Marfan syndromme is to say the least challenging. A multidisplinary approach is required. An orthopaedic specialist, a cardiologist, an ophthalmologist a geneticist, a radiographer and a specialist nurse are part of the team involved in the diagnosis of Marfan. Engaging this myriad of specialists can be costly to the sufferers of Marfan syndromme. To diagnose Marfan the entire FBN1 gene has to be screened. This process is expensive and is only available privately even in developed countries. Furthermore genetic testing has a success rate of 70-80% and cannot wholly exclude marfan's. Skeletal features of Marfan's syndromme can be difficult to define.. The graphs define upper and lower body ration are age dependent and are not commonly available. Moreover they only provide mean values without standard deviation making their interpretation challenging. The usefulness of span greater than height has been put into question in the diagnosis of marfan's syndromme. (Schott, 1992) the concept which was a trademark with Leonardo da Vinci allows man to be assessed or portrayed in a square. However anthropometric studies have revealed that span exceeds height by a value of 59-78% in the normal adult population. In addition assessing whether pes carinatum or pes excavatum is mild moderate or severe is difficult clinically. Similarly features that commonly lead to referral like a tall thin body are not included in the diagnostic criteria neither can they be used to discriminate against patients with marfan's syndromme. It is also important to note that the progress and appearance of symptoms of Marfan's over time makes the diagnosis difficult. All features do not appear at once and evolve over time from child hood to adolescence. There are conditions have clinically presented themselves like marfan's syndromme and have clinical diagnosis a difficulty. For Instance the MASS syndromme. (Glebsy and Pyeritz 1989) This disease presents with Mitral Valve Prolapse. This is a situation where the mitral valve of the heart closes properly but allows blood to regurgitate into the heart during ventricular contraction. The diameter of the Aortic root may be towards the upper limit of normal however there is no progression to an aneurysm neither is there development of aortic dissection. There are also Stretch marks of the skin that are unrelated to weight gain. Finally the disease presents with skeletal features of marfan's like scoliosis joint hyper mobility. MASS syndromme is a connective tissue disorder. It is inherited in an autosommal dominant pattern and is associated with mutations of Fibrillin 1 gene. FBN1. Family history may be positive however patients with MASS syndromme never develop dislocation of the lens or aortic root enlargement. Another condition is Stickler syndromme or Hereditary athroopthalmopathy (Stickler et al. 1965) This disorder is also multisystem. Its diagnosis requires the eye. Craniofacial and another system should be affected. Salient features of this disease include myopia degeneration of the retina and the vitreous, detachment of the retina, deafness athropathies, hyper mobility of the joints facial hypoplasia and microganathia. Most of these features are shared with Marfan's syndromme . In Shprintzen -- Goldberg syndromme (Shprintzen and Goldberg 1982) there are skeletal changes that are suggestive of Marfan syndromme. However these patients have craniosynostosis and neurodevelopmental abnormalities. In addition aortic dilatation may be present. Other conditions are Congenital Contractual Arachdinodactyly (Viljoen 1994) .Familial Aortic Dissection,( Nicod et al. 1989) Familial Ectopic lentis (Tsipouras et al. 1992) and Familial Marfan like Habitus ( Milewicz et al. 1995) In some countries or some clinical setting either the Ghent criteria or the Berlin Nosology is still employed in diagnosis. Prior to advancement of molecular biology diagnosis of Marfan's depended on the Berlin Nosology. However this criteria was not strict in its approach and at times lead to over diagnosis of patients with Marfan's syndromme. (Pereira et al. 1994) This was further emphasised by ( Rose et al. 2000) in this study 19% of patients who were diagnosed under the Berlin Nosology failed to pass the test of the Ghent nosology. In addition this study came up with an interesting finding. Dural ectasia was found to be the second most common major diagnostic manifestation. It was found out that screening for dural ectasia established the diagnosis of Marfan of syndromme in 23% of patients under the Ghent Nosology. Another glaring weakness of the Berlin Nosology was that it encouraged the diagnosis of Marfan using non-specific criteria once the diagnosis of Marfan was made in a first degree relative. In this scenario the only requirement for a diagnosis to be made was the identification of a trait commonly seen in Marfan patients in two organ systems. Problems of over diagnosis and Misdiagnosis arose. The present proposal is more stringent in that, in addition to a family history of Marfan syndrome (which still means identification of an individual in the family who independently satisfies diagnostic criteria on clinical grounds alone), the presence of a major clinical manifestation and involvement of a second system are required for definitive diagnosis. The Ghent criteria aims to be as objective as possible in selecting those with Marfan's syndromme. However several shortcomings have been noted. For instance most manifestations are age related and some as earlier discussed are difficult to quantify. In addition few studies have come up to test the sensitivity and specificity of Marfan's syndromme anomalies. For example only recently has the association between dural ectasia and Marfan syndromme been pointed out. (Pyeritz et al. 1981) Little is Known about the Prevalence of dural ectasia and further studies are therefore needed to qualify this presentation as a major criteria. There are also psychological issues that one has to undergo through when they are labelled to Have Marfan syndromme. Read the full article

[nnn-res-blog]

Effects of multidisplinary team based nurse-led transitional care on clinical outcomes and quality of life in patients with ankylosing spondylitis

To investigate the impact of transitional care by a nurse-led multidisciplinary team on clinical outcomes and quality of life of patients with ankylosing spondylitis. Read more from Asian Nursing Research https://www.asian-nursingresearch.com/article/S1976-1317(18)30545-0/fulltext?rss=yes

[evoldir]

Fwd: Postdoc: Valencia.SARS-CoV-2_Evolutionary

Begin forwarded message: > From: evoldir@evol.biology.mcmaster.ca > Subject: Postdoc: Valencia.SARS-CoV-2_Evolutionary > Date: 7 May 2021 at 06:51:28 BST > To: rdmpage@gmail.com > > > > BIOINFORMATICS POSTDOC IN SARS-CoV-2 EVOLUTIONARY GENOMICS > > Institute for Integrative Systems Biology (I2SysBio), UV-CSIC, Valencia, > Spain > > A postdoctoral position (1.5 years) is available immediately in > the PathoGenOmics group of Mireia Coscolla in Valencia (Spain) > www.uv.es/pathogenomic > > We are seeking enthusiastic applicants with skills in computational > biology/bioinformatics and with experience in data mining and comparative > or evolutionary genome analyses. Skills in Bayesian phylogenetics and > phylodynamics, and pathogen evolution is a plus. > > The project: Strategic initiative for genomic surveillance and assessment > of the impact of SARS-CoV-2 mutations in real time. The aim of the > position is to investigate molecular epidemiology dynamics, mutations > surveillance, and to collaborate with a multidisplinary team to explore > the functional impact of the different SARS-CoV2 variants. The project > is highly linked to the ongoing project https://ift.tt/34AyKAj. > > The tasks of the postdoc will be in sequence and evolutionary analyses > of SARS-CoV-2 sequences within a consortium with eight research groups > ranging from virology to mathematics and protein structure.  The position > is based in Valencia, at the Institute for Integrative Systems Biology > (I2SysBio), a mixed institute between university of Valencia and > CSIC. The Pathogenomics group is part of the Pathogen Systems Biology > program, and we study microbial pathogens in the context of their host and > disease. To approach this, we use a range of different omics technologies > where genomics is our main approach to study the evolution and molecular > epidemiology of the pathogen. But we also perform experimental infections > to discover which genomic determinants are involved in different virulence > readouts, using among other approaches transcriptomic of host and pathogen > To apply send CV and e-mail of two references to Mireia.coscolla@csic.es > > > Mireia Coscollá Devís: > www.mireiacoscolla.com > Principal investigator: > www.uv.es/pathogenomic > I2SYSBIO, Parc Cientific - Universitat de València > C/Agustín Escardino, 9, 46980 Paterna (Valencia) > Telephone.: 0034 963543317 > e-mail: > mireia.coscolla@uv.es > > > > Mireia > via IFTTT

[aworkstation]

The housing market, as we all know, is sort of, slightly, maybe, a little bit…well, screwed. In major cities, at least. Amanda Lwin, curator of Unreal Estates, a group show which opens in an estate agent’s office in east London…

The post New multidisplinary exhibition set in an estate agents explores the housing crisis appeared first on AWorkstation.com.

[thefranklinoutdoor]

Butter and Tin Jordan Martins

Opening: Sunday, September 9 from 2-4PM On view from September 9 to November 17, 2018 In Partnership with EXPOCHGO, extended hours: 

Friday, September 28 from 6-9PM

https://www.expochicago.com/visit/expo-art-week/gallery-exhibitions

Butter and Tin (Cloaking Device #3) is an immersive installation by Jordan Martins that ponders enclosed spaces designed for heightened visual experiences, transforming the structure of The Franklin into a hybrid of a hunting blind, a shamanic hut, and a camera obsura. Each of these spaces function as a place to go “inside” in order to perceive or let in some outside landscape or distant experience. Building off of The Franklin’s existing inside/outside dynamic, Butter and Tin explores the ways in which this state of heightened vision overlap through their structural and conceptual frameworks, creating a hallucinatory space for both mundane and mystical experiences.

Butter and Tin is inspired by a passage in the 13th century Icelandic Vatnsdaela Saga, which describes an account of a viking man in Norway, Ingimundr, who is told by a Sami fortune teller that his destiny is to move to Iceland. To prove it, she tells him that a lost amulet of his is already embedded in a hillside there, and Ingimundr hires three Samis to travel to Iceland to verify her claims.

Ingimund said that he wanted to make a bargain with them – ‘I will give you butter and tin, and you are to undertake an errand for me in Iceland and search for my amulet and report back to me about the lie of the land.’

They answered ‘This is a hazardous mission for Sami messengers to undertake, but in response to your request we want to make an attempt. You must now shut us up together in a hut and our names must not be revealed.’ This was duly done. And when three nights had passed, Ingimund went to them.

Upon emerging from their hut, the Sami’s bluntly tell Ingimund that they had traveled to Iceland and the fortune was true. The reader is given no information as to how they traveled across the ocean from a small hut, whether it was a physical teleportation or astral projection. Which raises some questions:  were butter and tin used purely for functional purposes, or for some trancendental properties that facilitate teleportation to other lands? Did they simply lock themselves in the shed, eat butter, tell jokes and upon emerging tell made up stories to their client? In his installation, Martins ponders the full spectrum of explanations, creating a space for beer drinking, mystical contemplation, and everything in between. Rather than pointed a distant mysterious landscape, the artists studio itself is invoked as a site of becoming where metaphysical and worldly goals co-mingle.

Jordan Martins is a Chicago based visual artist, curator, educator, and musician. He received his MFA in visual arts from the Universidade Federal da Bahia in Salvador, Brazil and is currently a lecturer at the School of the Art Institute of Chicago and North Park University.  He has run programming at Comfort Station since 2011 and has served as Executive Director of Comfort Station since 2015. Martins’s visual work is based in collage processes, including mixed media two dimensional work, photography, video and installation, and he has exhibited nationally and internationally. He is co-director of the Perto da Lá, a biennial multidisplinary art event with international artists in Salvador, Brazil.  From 2014 to 2016 he served on the programming committee for the Chicago Jazz Festival. He was a resident in the Chicago Artists Coalition’s HATCH program in 2013, a mentor for their LAUNCH program in 2016 and 2017, and currently serves on their Educational Advisory Panel. In additional to his involvement with the exhibition programming at Comfort Station, he curates the visual art program at Elastic Arts.