#genetically correct
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Got tired of using simpe every time i start a new save/uberhood (which is much more than i'd admit) so i've made my own version of strangetown! this uses @average-vidcund-enjoyer's V2 gc strangetown premades on Dorsal Axe's Fixed Strangetown V 1.3. i've also moved in the Singles and Ajay Loner into their lots (which come in with the essential furnishings and nothing else) for my conveinence. Download here (Main Hood) Download here (Shopping District)
#sims 2 download#ts2cc#s2cc#sims 2 strangetown#strangetown#sims 2 neighborhood#genetically correct#sims 2 genetically correct hood#sims 2 gc hood
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The Brokes, but genetically correct
loooooonnng picture-heavy post ahead :)
Skip to the download: CC-free Sims2Packs + .simpe face templates
Hello all! It's been awhile...I know. Life is life.
I do still love making genetically correct sims and recently I've been wanting to start a new Pleasantview. I like my old genetically correct Pleasantview, but it was a first try and I'd like to give it a bit of a refresh. Plus it's fun for me. :P
This time around I want to slowly work on each family + lots, releasing them slowly at first and then have the combined hood at the end. This allows me to really focus and expand on each one. Plus I'm generally not very motivated, but I think working small parts at a time will help this. All parents extracted from Croconaw/MeetMetotheRiver's Clean Pleasantview template. Offspring created in CAS.
A little refresher on the Newbie-Broke lineage...
To note I only considered the genetics of the previous generation in making the current playables. For example, Betty Newbie's appearance was extracted directly from SimPe. She was not genetically created with recreated Vadim/Fatima Simovitch. I did attempt this, but found that Brandi would look too dissimilar from her original non-gc face. I still want to keep with the general aesthetics of the original faces, just obviously improved, especially for the second-gen playables like Dustin and Beau.
So first, we have the Newbies:
Bob + Betty = Brandi
slight face shape changes, smaller less-bug eyes
The Brokes:
Flat + Flo = Skip, Ruby
smaller Broke nose, forehead/brows from Flo
Bonus! Ruby Broke, Skip's older sister seen in TS3.
"Ruby Broke is thinking about moving out but she enjoys the convenience of having family close by."
I absolutely LOVE her face. I think it's a perfect mix between Flat and Flo. I did garner inspiration for her hair/outfit/eyes from her Sims 3 version.
And finally...the boys.
Brandi + Skip = Dustin, Beau, Unborn Baby Broke
Dustin -
looks completely different, less sad and looks far more like Skip
Beau -
again looks very different...but now actually looks related to Dustin, yet not like twins which I thought I lacked on last time.
and there we are...
I have both the individual sims downloads, a combined package, plus the .simpe face templates that you can use to replace in any Pleasantview or other hood right now
Note: Female sims will take your default replacement makeup or lack thereof. So warning they make look a little bit crazy at first.
Credits:
Tutorial on Facial Structure Changes
Tutorial on how to extract in-game sims
DOWNLOAD
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GENETICALLY CORRECT STRANGETOWN using carrit's recreation of ancestors

Genetically correct Strangetown is here and it couldn't be any stranger.
I couldn't have made this without carrit's recreation of Strangetown's ancestors.
If you want my Pleasantview version, click here!
What you will need : Creature fix (for Jill's alien eyes to function correctly)
As you can see, Pascal and Lazlo weren't given any changes, simply because they were already genetically correct.
BONUS : Jill and Buck as adults.

PICK AND CHOOSE : Simsfileshare | Dropbox
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Is the gene that makes housecats black/solid the same gene that makes panthers black?
Depends on the species! Leopards have a recessive ASIP mutation just like housecats, but jaguars have a dominant MC1R mutation, the same gene that makes yellow/red variants in a lot of animals. Gain of function MC1R mutations are (co)dominant and make the animal produce more eumelanin, loss of function mutations are recessive and result in more phaeomelanin. (For ASIP, it's the opposite.)
Both genes are responsible for melanism in several felids:
ASIP: domestic cat, leopard, asian golden cat, pampas cat, kodkod
MC1R: jaguar, jaguarundi*, geoffroy's cat
*This one is interesting, because the reddish variant seems to be the original and the more common dark is the later mutation.
And the species with still unknown genetics: serval, oncilla, marbled cat, bobcat, southern tigrina, margay, jungle cat, african golden cat, african wild cat**
**Technically i think it's possible that the melanism of domestic cats is a direct heritage of the african wildcats from before domestication OR that the melanism of african wildcats is an evidence of crossbreeding with domestic cats; either way they would have the same ASIP mutation.
#i always instinctively interpret 'panther' as 'leopard' first because in my language the similar 'párduc' refers exclusively to leopards#and i have to manually initiate that No This is A Different Language; panther=jaguar or even =puma is Correct Here#ask and answer#cat genetics#other animals#agouti#extension#i'm an influencer now
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nobody needs to be above 5’5 so if you are, then saw off those extra inches immediately. and if you are taller than me, you will have to kneel when you address me. i will not repeat myself. so spread the word before i start killing people. many thanks
#ro talks#my life is hard enough but on top of everything else going on i’m also 5’3#like jesus i have lost the genetic lottery in every possible way#but i refuse to lose any longer. you will all simply have to take my word for law#no arguments allowed. remember i am always correct and also always in charge
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Are you perhaps going to add Bill's evident medical trauma into the story?
Bill's been getting called insane for seeing the third dimension by his mom since chapter 15 and I've mentioned his vision is tied to a medical condition he was born with. We were already going there.
Only difference is now he's going to an optometrist in addition to an orthopedic doctor.
#(I forget if he's directly mentioned he has a medical condition in the fic yet or just danced around it)#(99 percent sure he's said this vision is something you're either born with or not)#(anyway I've written a ton of out of fic posts about his medical condition—possible genetic origins—how it works—how it's seen culturally—)#(so yeah this has been the plan since. probably like May last year?? May or June?)#(since before I wrote the post on non Euclidean new age cults)#(if his homeworld actually is named Euclidia calling his geometry non Euclidean is gonna retroactively be so funny)#(it's the correct term but that makes it funnier)#anonymous#ask#bill goldilocks cipher#the book of bill spoilers
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I think my favorite thing about doing ginger red hair instead of cherry red hair is: lying to people about it
#I love the cherry red / wine red and I'll probably go back at some point bc it's my Origin.#but for now.#I don't actively lie to people but bc it's a Natural and Plausible hair color#and I'm already pale and I dye my eyebrows to match my hair. ppl figure it's natural#and it has come up MULTIPLE TIMES. and I've recently been rolling with it instead of correcting ppl. bc who cares?#recent examples that come to mind (but I did correct them in this one) my surgeon assuming it was natural#and using my genetics as a natural redhead as a baseline to tell me about what I can expect from my future scarring#and then again later with the anesthesia. they were going to dose me differently#the anesthesiologist glanced at me when I came into the OR and was getting the stuff ready on his cart#and when he heard me talking to my doc and re-telling him that oh the hair isn't natural#he was behind the curtain like FUCK#taking shit off his cart and quietly redoing his setup#that's how I learned that redheads need higher doses of anesthesia than other ppl.#they also need more of the topical stuff like lidocaine. apparently they metabolize it faster(?)#ANYWAY he was going to up my dose thinking I needed it lol#so i almost got way more sedatives and pain meds than i needed bc of my hair dye LMAOOO#other more Normal Life examples was a country dude in full hunting gear holding a door open for me someplace#and I said thank you and he lifted his hat up to point at his (natural) red hair and said ''twins!''#this one sticks with me because that was such a cute thing to do. what the hell#and at snakefest I was talking to some people at their food truck. there was an older guy who trapped me into a convo for like 30mins#he was Very Nice. and they were going to some type of irish festival next and said I should go too bc I'll be right at home#flat out just was like. this bitch looks irish#and I don't know why all of this is so funny to me. it has no reason to be.
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I think it’s cute if lemongrab can go to wizard city, like the wall lets him in and he's never thought about it before or questioned it, but he doesn’t really exactly grok that he’s A Wizard
he knows that he doesn’t get in trouble for going there, but he knows that some people do. which is kind of a big deal for him
#adventure time#wizard city#<- cuz larry#lemongrab#larry#digital#being a wizard is really more of a molecular thing than an education/trained-in thing#you’re deeply sad and mad and magic glued itself onto your cells bc of it? OK! the wall will let you in#you’re genetically predisposed to magic and also it’s tearing you apart mentally to be able to see the spirit realm and stuff?#OK! the wall will let you in#the wall clearly isn’t trained on each specific wizard. because there’s a SCHOOL OF NEW UNTRAINED CHILDREN#who could all get in. unless they all had to go to an early orientation to teach the wall their voice so they could enter the city#insane.#wc and its population of baby wizards is a menace to ME SPECIFICALLY#okay wow that was all unrelated.#i do think lemongrab would have a problem with the password.#some kindly wizard would be like oh sure young lad password's 'wizards rule' and lg would be like Excuse Me.#wizards rule WHAT. (wall disappears)#and the other guy'd be like well like they just rule in general. dont you agree. raises eyebrow. ahem. young wizard.#and lg would be like ummm i think they need to earn that kind of respeckt. >:/#he begrudgingly agrees it's OK because they 'rule' the city. so technically it is correct. but he has middling problems with it#as a guy who cares about saying correct statements and being precise
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Goodbye finals, hello new Pleasantview!
#omg I haven't played in so long#playing genetically correct pleasantview this time#lets hope my save doesn't just die again!!#ts2#the sims 2#simblr#sims 2#sims 2 screenshots#ts2 gameplay#ts2 pictures#ts2 simblr#pleasantview 2.0#don lothario#nina caliente#Lothario round 1
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Kekkei Genkai are a recessive or dominant trait? It is possible that someone born from two parents without Kekkei Genkai in a clan that have one, inherit the characteristic?
Kekkei Genkai Genetics
Good question. Unfortunately, I did not study biology, but I did do some research (mostly asking some biology-savvy people in the Naruto fandom and digging out my 10th-12th grade biology book - they did not, however, proofread this current version of the post). Still, take everything here with a barrel of salt unless you see this post getting peer-reviewed.
What you are asking is whether or not, in the world of Kekkei Genkai, parents can be "hereditary carriers" as opposed to "users" of a Kekkei Genkai gene. To refresh everybody's minds, this usually occurs in recessive inheritance patterns. However, spoiler alert, I think a lot of Kekkei Genkai don't rely on recessiveness and dominance alone and, technically, a person can also end up as a hereditary carrier of a dominant gene. It's not exactly the same concept but results in a similar situation.
(I hope you don't mind me explaining irl biology first; if you do, skip the first half of this post until you reach the Sharingan section)
Recessive and Dominant Inheritance Patterns
Recessive or dominant inheritance occurs because genes always come in double packs - one half of a child's chromosomes originates from the mother and the other half originates from the father. However, those two gene variants (also referred to as "alleles") do not always contain the same genetic information. This is when a child will end up with a "heterozygous genotype".
As Anon previously mentioned, one of the two heterozygous alleles will usually play a recessive role in relationship to a dominant allele, the latter of which will "win" against the other and determine the phenotype.
For communication purposes, we will always assign each allele a letter. Dominant alleles use upper case letters (eg. "B") and recessive alleles use lower case letters (eg. "b").
To visualize this, if one parent has brown eyes and contributes a dominant (B) gene while the other parent has blue eyes and contributes a recessive (b) gene, the child will end up with a heterozygous genotype (Bb), that contains both genes.
Since the allele for brown eyes (B) dominates the blue eyes allele (b), the child will have brown eyes as a phenotype. The child remains a carrier of the recessive blue eyes allele (b), however. Imagine it like a smaller gene being hidden behind a bigger one. The recessive allele is still there, you simply just cannot see it.
If a child instead inherits two blue eye alleles (b) from their parents, they will be born with a homozygous genotype (bb), translating into blue eyes. This is the only circumstance under which a recessive gene won't be hindered by a dominant one.
Additionally, a genetic trait itself is never inherently recessive or dominant. "Recessive" and "dominant" merely describe the relationship between two alleles. A gene that is recessive toward one gene can still be dominant in relation to another (eg. brown eyes > green eyes > blue eyes).
To visualize: A Chunin might dominate a battle against a Genin but a Jonin will still likely wipe the floor with that very same Chunin. Same applies to dominant inheritance patterns.
TL;DR Dominant traits (B) are always expressed. Recessive genes (b) are only expressed if no dominant gene is present. Dominant (B) + Recessive (b) -> Dominant Inheritance (Bb) Dominant (B) + Dominant (B) -> Dominant Inheritance (BB) Recessive (b) + Recessive (b) -> Recessive Inheritance (bb) "Dominant" and "recessive" are not absolute as they don't define the genes themselves but their interaction with another gene instead. A trait (g) dominant to a recessive trait (b) can still be recessive to a different trait (B). b < g < B
The Limits of Dominance and Recessiveness
The problem is, those previously established rules surrounding recessiveness and dominance do not always apply. Two well-known examples are co-dominance (eg. blood types) and incomplete dominance (eg. red + white = pink flowers) in which neither allele in a heterozygous genotype is recessive and instead results in an entirely new phenotype.
Not just that, dominant/recessive gene inheritance patterns are not the be-all and end-all of gene inheritance and are sometimes mixed with and influenced by polygenic, epistatic, or gonosomal inheritance patterns.
Polygenic Inheritance
In the real world, polygenic inheritance is common in traits like skin color and height. In the world of Naruto, it might come into play for traits like nature affinity or chakra potency.
If a polygenic inheritance pattern is present, the trait will be determined not by a yes/no question but instead by the cumulative effort of multiple genes. It will create a spectrum of phenotypes, depending on both the number and the exact genes that have been inherited.
To visualize the concept, imagine dropping food dye into a glass of water. The more drops of food dye and the bigger the drops of food dye are, the stronger the color inside the glass becomes.
In reference to Anon's ask: If a Kekkei Genkai were to be polygenic, parents might pass on a Kekkei Genkai to a child in spite of not having access to the ability themselves, simply just because their child reached a certain "threshold" whereas the parents did not (eg. your water nature affinity is not strong enough for you to acquire ice release). This can be entirely independent of "gene dominance".
Epistasis
Epistasis, similar to polygenic inheritance (as well as co-dominance and incomplete dominance), describes the interaction of two or more non-allelic genes. Unlike polygenic inheritance, epistasis produces an entirely new phenotype rather than a spectrum of phenotypes.
Epistasis comes in various forms, hence coming up with just one analogy wouldn't be doing it any justice. Sometimes, one gene masks the effect of another (eg. a person without hair cannot be blonde), and sometimes, genes instead require one another for a phenotype to go into effect (eg. sweet pea flowers will be purple but only if two specific mutations interact - otherwise, they will be white). In Naruto, this may play a role in the inheritance of the Rinnegan among others.
In reference to Anon's ask: Just like before, this can easily create a situation similar to the traditional hereditary carrier. A parent may carry a gene that they themselves have no access to as it either requires the presence or absence of yet another gene.
Gonosomal Inheritance
Additionally, as opposed to being located on one of the 22 different autosomal chromosome pairs, genes can also be passed on through gonosomes (X and Y chromosomes) and therefore rely on the gender of the child.
Traits passed down via the father's Y Chromosome will always be present in his sons, never in his daughters. If it's via the father's X Chromosome, it will never be inherited by his sons, only his daughters. If it's on the mother's X Chromosome, sons have a 50% chance of inheriting the gene. For girls, this number is entirely reliant on recessiveness/dominance.
In reference to Anon's ask: If a Kekkei Genkai were to be gonosomal, the father could never be a "mere" carrier and would typically have access to the ability.
In-Universe Examples
Now, there are multiple different Kekkei Genkai in the world of Naruto, all of which are determined by different, non-allelic genes entirely (otherwise, individuals like Kaguya or Mei Terumi would not have more than one Kekkei Genkai). This means that we cannot make blanket judgments about all Kekkei Genkai when it comes to their respective inheritance patterns - We can only make case studies.
However, any resemblance to the previously mentioned inheritance patterns might just be coincidental after all. Kishimoto's writing itself doesn't give me much of a basis when trying to gauge the extent of his expertise in genetics. Honestly, that's just how wild genetics can be, giving a biological basis even to the random whims of an author.
Regardless, I'd like to go through a couple examples.
Example: The Sharingan
Since I usually discuss Uchihas, let's use the Sharingan as our first example and apply a purely dominant/recessive inheritance pattern both for simplicity's sake as well as to better accommodate Anon's ask.
For communication purposes: E = Dominant Sharingan Gene e = Recessive Sharingan Gene N = Dominant Non-Sharingan Gene n = Recessive Non-Sharingan Gene
Sasuke and Sakura have a daughter, Sarada, who has inherited the Sharingan. As far as we know, Sakura does not descend from the Uchiha Clan, which means that she also doesn't carry the genetic information for the Sharingan on either allele. In other words, Sasuke is the only parent whose genes contributed to the manifestation of Sarada's Sharingan.
This outcome can only arise under the condition that at least one of Sasuke's alleles is dominant (En or EE) in relation to Sakura's. So, depending on whether Sasuke's genotype is homo- or heterozygous, these are the two most probable possibilities:
Option 1: Sasuke's genotype is homozygous and dominant (EE)
Option 2: Sasuke's genotype is heterozygous and has a non-Sharingan gene (En)
The two most probable scenarios aside, even supposedly dominant/recessive inheritance patterns can be more complicated than that.
For example, the Sharingan may also be inheritable via two different genes (E and e) of varying dominance. Hypothetically, this may either occur through the introduction of another Otsutsuki Clan member's genes or the mutation of the initial Sharingan gene that originated from Indra.
This could give Sasuke a heterozygous genotype (Ee), passing on different Sharingan genes to his children, as you can see in "Option 3". Unlike previously, this might cause Sasuke's children to be born as hereditary carriers (individuals who have inherited the genes but do not express the trait due to the gene's recessiveness).
Option 3: Sasuke's genotype is heterozygous (Ee)
But all of this only applies under the condition that Sakura's alleles are homozygous.
Similarly to "Option 3", there's an "Option 4" in which there are multiple different "non-Sharingan" genes in the Naruto-verse. Some of these non-Sharingan genes may be dominant (N) and some others might be recessive (n) in relation to the Sharingan gene (E). Under this condition, both NE genotypes (the Sharingan is not expressed), as well as En genotypes (the Sharingan is expressed), may occur.
Option 4: Sakura's genotype is heterozygous (Nn)
In yet another scenario, Sakura may have Uchiha ancestors and secretly carry the Sharingan gene. This may hypothetically be the case if one (or more) of Sakura's ancestors had been married to or had had an affair with an Uchiha. Since Sakura herself never awakened the Sharingan, we have to assume the gene that was contributed by Sakura's Uchiha ancestor is recessive (e).
Option 5: Sakura's genotype is heterozygous and has a Sharingan gene (Ne)
And, of course, there are multiple combinations of all of these previously outlined options. I won't explain them all here, but you get the gist.
That being said, the Sharingan being a "generally more dominant" trait remains the more probable option here.
While Sakura having a secret recessive Sharingan gene (e) is theoretically possible, it would be a very unlikely coincidence of secret clan ancestries. It is much more likely that Sarada's genotype is heterozygous (En). In addition, we know for a fact that Indra Otsutsuki's genotype was heterozygous (En) as well. Since she couldn't possibly have possessed the Sharingan, Indra's mother must have contributed a recessive gene. So, if nothing else, there is at least one recessive non-Sharingan gene in circulation.
While the Sharingan itself is still supposedly rare within the Uchiha Clan, this could instead be the result of "low penetrance", as @the-real-sasuke-uchiha suggested. The obvious threshold here is the Sharingan's awakening condition in the form of psychological trauma and/or turmoil, constituting an environmental factor. Psychological responses, in turn, can also be influenced by genes that influence a person's response or resistance to stress, anxiety, and trauma.
Example: The Byakugan
I won't go as far into the details as last time, but the previously illustrated Sharingan logic also applies to the Byakugan.
In the case of the Byakugan (b or B), we know that only one of Hinata's children inherited the Dojutsu (assuming this is not the result of low penetrance instead). Boruto got a recessive allele from Hinata (b or n), whereas Himawari received a dominant allele. For this, either Naruto and/or Hinata must have a heterozygous genotype.
Hierarchy: N > b > n B > N > b
Naruto is heterozygous (Nn): Boruto (Nb, nn, or Nn) = Naruto (Nn) + Hinata (bb or bn) Himawari (bn) = Naruto (Nn) + Hinata (bb or bn) Naruto is homozygous (nn): Boruto (nn) = Naruto (nn) + Hinata (bn) Himawari (bn) = Naruto (nn) + Hinata (bn)
But there's also a hypothetical scenario in which the Byakugan is a recessive trait and Naruto has a secret Byakugan allele.
Boruto (Nb) = Naruto (Nb) + Hinata (bb) Himawari (bb) = Naruto (Nb) + Hinata (bb)
And just like last time, you need to consider that there might instead be two different Byakugan alleles of varying "dominance" in circulation (B and b).
Boruto (Nb) = Naruto (NN) + Hinata (Bb) Himawari (BN) = Naruto (NN) + Hinata (Bb)
And of course, then there's also the possibility that Naruto's and Hinata's genes are co-dominant on the "Byakugan allele". This might have expressed itself through Himawari's blue eyes that only turn white whenever the Byakugan is activated. Alternatively, this is just a regular eye pigmentation gene, unrelated to the Byakugan.
Example: Nature Combinations
While Earth and Water Release are the basis of Wood Release, in the manga, Kakashi points out that their simultaneous activation is what defines the Kekkei Genkai, meaning that the presence of two basic chakra nature affinities is non-sufficient. This means that the inheritance of nature combinations is both influenced
By a polygenic collection of genes determining your individual affinities for the five basic chakra natures (Earth, Water, Fire, Wind, Lightning), which are the basis for the Kekkei Genkai.
By an additional gene that determines your ability to activate two or more chakra natures simultaneously and combine them, probably constituting epistasis. However, without affinity for two or more basic chakra natures as a basis, the gene would be masked (read: useless).
Of course, this doesn't mean that dominant and recessive inheritance patterns don't play a role. Both the nature affinity as well as the Kekkei Genkai genes are likely subject to recessive/dominant inheritance patterns.
As for a real-world example, let's look at the inheritance of coat color in mice. It follows both an inheritance pattern of epistasis as well as one of recessiveness/dominance. The "black coat" gene is dominant toward the "brown coat" gene. However, there is also a dominant "pigment" gene and a recessive "no pigment" gene at play. The resulting black, brown and white mice are a complex interplay between their parents pigmentation and coat color genes.
Now, if we apply this to Haku and his Kekkei Genkai, we can assume that he inherited a dominant Ice Nature Combination gene (I) from his mother (compare with Chapter #29). That's because his father should naturally lack the gene unless he has secret ancestry from the Yuki Clan (technically possible but unlikely).
Haku (In) = Mother (II or In) + Father (nn)
But it is also entirely possible that Haku's mother could not use Ice Release herself, due to lacking Wind (A) and/or Water (B) Release (or both). That would make Haku's mother more of a "carrier" of the gene rather than a user, meaning that her husband would have contributed the genes for Haku's nature affinities.
Now, unlike Ice Release, Hashirama's Wood Release might be recessive instead as none of his relatives have ever been said to possess the Kekkei Genkai and there is no known association with the Senju Clan as a whole. Again, penetration plays a role, but we have not been given a reason to assume that Wood Release relies on some sort of trigger.
Back to Anon
Now, this was a whole lot of needless fantasy science but it was fun regardless. I hope I could give you an approximate idea on just how complicated genetics are how inheritance patterns might affect the distribution of Kekkei Genkai within a blood lineage.
Regarding your initial question, Anon, there are no definitive answers, unfortunately, for multiple reasons:
"Recessive" and "Dominant" are not absolute and depend from gene interaction to gene interaction. Heck, there might even be different versions of the same Kekkei Genkai gene.
Different Kekkei Genkai may not (and most likely don't) follow the same inheritance patterns as other Kekkei Genkai.
While we can make educated guesses, we don't know the definitive inheritance patterns (polygenic, epistatic, gonosomal, etc.) of any Kekkei Genkai. From an irl POV, we don't even know whether Kishimoto had any thoughts on the matter.
But once again, recessive/dominant inheritance patterns are not the only setup that can lead to surprise Kekkei Genkai users born to parents without Kekkei Genkai abilities. Nature Combinations almost certainly do lead to such as they most likely rely on epistasis (unless, I don't know, the Ice Release gene always comes in a triple pack with Water and Wind release). The Byakugan might just meet the criteria for a recessive inheritance pattern (unless Hinata's genotype is heterozygous) and even if it isn't, it could be of low penetrance instead. Meanwhile, the Sharingan appears to be mostly dominant.
#naruto#naruto analysis#naruto meta#naruto discussion#kekkei genkai#sharingan#byakugan#ice release#ask#naruto ask#anon ask#anon#i initially wrote this post multiple months ago because i suddenly fixated on fantasy genetics 😭#and then edited it sooo many times#aaahhhh#hopefully i actually understood this shit correctly#anyway please keep in mind this didn't get peer-reviewed :') yet#the other tumblr users that i kept bothering are free to review and correct me and i'll go and edit the post 🙈
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Quickly realizing why no one makes genetically correct Veronaville sims starting from generation one, Cleopatra's children are actual body horror. That being said I'm happy with how Contessa and Consort are coming along
#They need to look 200x snootier but i like Contessa's nose#Consort does not look like himself it's uncanny#Early Veronaville musing#Genetically correct premades#For context: Early PV and ST both come with genetically correct variations so I hope to do the same for Veronaville#Though I made a Cordelia a while back that I’m really happy with so I might make Consort look more like her
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Is calico a possible Creasia coat color?
Ah, such a simple question, but so complicated to answer! Is it possible? Maybe. Likely to be expressed? No. You probably know how calicos are produced and anyone who doesn't can google that. Short review: you need at least one of the color alleles (in the case of the domestic cat, the black/red) located on the X chromosome. So males are always homozygous, but females can be heterozygous. Then you need the thing where neither color is dominant. Instead, females express both colors. This event alone will produce a tortie, not a calico. To get calico, you need one more thing: the piebald gene, which is present in many animals. It produces random colorless white patches, interspersed with the underlying color(s). Finally, in order to get a classic calico, you need the female zygote to have discarded her extra Xs (turned them into bar bodies) early enough to produce solid patches instead of a more blended coat. NOW you have a classic mi-ke calico. She will almost always be female, but can be male XXY or male chimera (having absorbed his twin!) or possibly an even rarer genetic event. On the flip side, solid red or black cats are male something like 75% of the time. Female solids would be homozygous for the trait, which is rarer. There are other genes that effect cat coat color and pattern, and of course there can always be new mutations, but those are the basics. Question: is color a sex-linked gene in other species of cats? Big cats have many things in common with domestic cats. Is this one of them? I could not find an answer to this. Probably no. But... I did have Caraca say at one point when she and Halvery were trying to reproduce Roup's color, "Solids are more likely to be male." This strongly suggests that some aspect of creasia color is on the X. So even if this is unlikely for big cats in our world, I have sort of committed to it on Lidian. But... Is the piebald gene present in other cats besides domestic cats? Again, no answer. The closest I could find was the white band gene that produces golden tabby tigers. It is not actually piebald, though. It's a completely different gene. If you read the wikipedia article about golden tabby tigers, you will notice a lot of inbreeding. This is how most usual colors in domestic (and semi-domestic) animals get a foothold. People notice a single unique mutation, decide they love it, and do some aggressive inbreeding to produce homozygous individuals. Many domestic animals have very shallow gene pools. Obviously, most of them handle it just fine. Wild animals avoid inbreeding. Nature tends towards outbreeding. Maximizing genetic diversity is a survival strategy, so animals evolve various barriers to inbreeding. There ARE more color variations in big cats that live in jungles/forests vs open plains. Tigers and leopards have more color variations than African lions. It doesn't seem to affect their odds of survival as much. Creasia live in dense forest, so it makes sense that they have some color variation. Rare mutations would pop up, but they would quickly disappear unless the creasia started selecting for them. So if they decided that piebald was fabulously beautiful and everyone wanted to have babies with those cats, they might quickly develop a piebald trait that is regularly expressed. But they tend to like colors that are "good for hunting," and those are browns/grays/black. They are starting to value other traits and expand their tastes, though. Anyway - the potential for something like a calico is probably in there somewhere. To specifically get calico, I think you'd see piebald popping up first.
#hunters unlucky#creasia#color genetics#color genetics in cats#anyone more knowledgeable is welcome to correct me if I have made mistakes
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ohh for the genetically accurate cats, barley or millie? thank you!!! ^_^


barley ll, XoY, Bbl, DD, dmdm, aa, Tata, WsWs millie LL, XoXo, bb, dd, dmdm, AA, McMc, Wsw wanna request a cat? see here: link + barley's family


jumper & hoot ll, XoY, Bbl, DD, dmdm, aa, Tata, WsWs violet ll, XoXo, bbl, DD, dmdm, Aa, Tata, Wsw wanna request a cat? see here: link
#i realized it'd be easier to get barley & his siblings to be genetically correct by drawing them all at the same time so#barley#millie#jumper#hoot#violet#warrior cats#wc#wc designs#ray's genetically accurate designs
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Genetically Correct Cassandra Goth
Download at Mediafire
And yes I have shared Cassandra in the past but she was wearing an Apartment Life hairstyle and I wanted to fix my mistake.
#sims2#thesims#thesims2#pleasantview#cassandra goth#genetically correct sims#sims 2 download#sims 2 makeover
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On my site: Cat breeding genes simulator
Use it here!
Note: The ginger gene is sex-linked, so only the XX parent (the queen) can be a tortie/calico in this simulation. But feel free to use this for gay cat pairings if you want to
#my art#furry#cat genetics#genetics#game#interactive#educational (?) i focused more on code than research#feel free to correct me if i got anything wrong#javascript#browser games (?)#stuff on my site
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Speculative carno colour variations. Second time drawing this species, I hope I did relatively well on the anatomy :thumbsup: Wanted to do some pattern variations too but I accidentally merged to many layers. Oops. Some design notes under the cut
-The most specific part of this design is around the eye area, featuring a tear streak to reduce sun glare, found in a variety of species today, including birds.
-The bright area around the eyes help with visual communication
-How much sight would be important to carno for evoloution to select for these patterns around the facial area is.... debatable. Their eyes are pretty unique but the area in their skull dedicated for the part of the brain processing visual signals does not appear to be very big. But theres no reason to not select for it as far as I am aware, and from a design perspective it helps drawing attention to their uniquely shaped heads and acts as a "landmark" for their pattern. -They take some mild inspiration from the Yellow Fronted Woodpecker. Both species is Argentian so I thought it would be fitting, hah. - I had some trouble in general figuring out how exactly draw these bump like structures described on carnos skin, but they were mentioned to have occured on their bodies in irregular patternless way so I tried paying attention to the randomness
#Z's art tag#carnotaurus#dinosaur art#speculative biology#I *think* that's the correct tag for this? It is speculative even though they arent my own alien species but an extinct one yk yk#amatuer paleoart#paleoart#carnotaurus art#speculative dinosaur genetics#art#I hope these are the correct tags#rare fandomless art post lol
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