#Metabolic Disorder

[xtruss]

Credit: Dana Smith

Understanding The Sudden Rise of Type 2 Diabetes In Children

The Metabolic Disorder Was Long Known as a Disease of Adulthood. Now, It’s Spiking in Kids and Teens, With Worrisome Consequences.

— By Charlotte Huff | July 31, 2024

The appearance of type 2 diabetes in children and teens puzzled physicians from the start. Fida Bacha recalls working as a pediatric endocrinology fellow in Pittsburgh shortly after 2000 when young, overweight and obese patients began to arrive at the clinic, some describing increased thirst, more frequent trips to the bathroom and other symptoms of what was then called adult-onset diabetes.

“It was a new realization that we are dealing with a disease that used to be only an adult disease that is now becoming a disease of childhood,” says Bacha, who practices at Texas Children’s Hospital in Houston.

More than two decades later, physicians and researchers are still trying to unravel what’s driving the emergence and proliferation of youth-onset disease, particularly among marginalized communities including Hispanics/Latinos. The increasing prevalence of obesity among young people is clearly one contributor, but researchers are also scrutinizing the potential influence of other lifestyle and environmental factors — everything from exposure to chronic stress and air pollution to sugar-rich diets. Along with physiological factors, such as where they carry excess fat, youths from lower socioeconomic levels may be vulnerable due to aspects of daily life beyond their control, such as more limited access to healthy food and opportunities to safely exercise in less-polluted neighborhoods.

As researchers try to sort out the interplay among genetics, metabolic factors and environmental influences in Hispanic and other populations, their goal is to answer this key question: Why do some seemingly at-risk adolescents progress to diabetes while others do not?

Long-term, the challenges and health stakes are significant. When type 2 diabetes first emerged in youths, clinicians initially thought its progression would mirror that in adults and thus could be treated accordingly. That hasn’t panned out, says Barbara Linder, a pediatric endocrinologist and senior advisor for childhood diabetes research at the National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK). For instance, researchers have determined that metformin, a commonly prescribed oral antidiabetic medication in adults, doesn’t work as well in young people.

“We know that the disease is very aggressive in youth and very difficult to treat,” Linder says. “So it’s really imperative that we develop effective approaches to prevention. And to do this we obviously need to be able to effectively identify which youth are at the highest risk.”

Even with treatment, young people develop other medical problems related to diabetes faster than adults, according to a study that followed 500 youths, more than one-third of them Hispanic. Sixty percent developed at least one complication within about 15 years after diagnosis, when just in their 20s.

“It’s really alarming,” says Luisa Rodriguez, a pediatric endocrinologist who studies type 2 diabetes and obesity in children at the University of Texas Health Science Center at San Antonio. For every 10 adolescents with youth-onset diabetes, she points out, “six of them, within a decade span, are going to develop a significant comorbidity that will highly impact their lifespan and quality of life.”

Complications of diabetes appear more quickly in young people than in older adults. Researchers studied 500 overweight adolescents, aged 10 to 17, who had been diagnosed with type 2 diabetes. Within 15 years of their diagnosis, 60 percent of the participants had developed at least one medical complication of diabetes, and 28 percent had developed two or more.

Insulin Resistance

In type 2 diabetes, the body struggles to use insulin effectively. This vital hormone, made by beta cells in the pancreas, helps glucose in the bloodstream enter cells in muscle, fat and the liver, where it’s used for energy. But sometimes those cells gradually lose their ability to respond to insulin, forcing the beta cells to pump out more and more of it. If the beta cells can’t keep up, blood glucose levels will begin to rise, leading to a diagnosis of prediabetes and, eventually, diabetes.

In the past, type 2 diabetes typically didn’t arise until well into adulthood. But now, cases in US youths ages 10 to 19 are rising fast. Since 2002-2003, overall diagnoses have doubled from 9 per 100,000 youths to 17.9 per 100,000 in 2017-2018, particularly among Asians, Pacific Islanders, Blacks and Hispanics. If those rising rates persist, the number of type 2 diabetes cases in young people is projected to skyrocket from 28,000 in 2017 to 220,000 by 2060.

Various factors have been linked to insulin resistance in childhood or adolescence, including obesity, inactivity and genetics, according to a review of the causes of type 2 diabetes in youths published in the 2022 Annual Review of Medicine. The disease tends to run in families regardless of race or ethnicity, which suggests that genes matter. Among US Hispanics, adults of Mexican or Puerto Rican heritage are most likely to be diagnosed, followed by Central and South Americans and Cubans.

Obesity is also a contributing factor: Slightly more than one-fourth of Hispanic youths are obese, a higher percentage than for any other major racial or ethnic group. Children also are more likely to develop type 2 diabetes if their mother has the disease or developed gestational diabetes during pregnancy. One theory is that fetal exposure to maternal diabetes while in the womb can spur metabolic changes following birth.

Puberty is also highly influential — most cases are diagnosed after its onset. During puberty, youths temporarily experience insulin resistance, due in large part to an increase in hormones, Linder says. Most youths offset that transient resistance by secreting more insulin, she says. But for reasons that are still unclear, a subpopulation of adolescents does not. “When they’re faced with this stress test of puberty, they can’t increase their insulin secretion enough to compensate,” Linder says. “And that’s probably why they develop type 2 diabetes.”

One analysis, which looked at type 2 diabetes trends from 2002 to 2018, identified the peak age for diagnosis as 16 years in boys and girls. The sole exception involved Black youths, in whom diagnoses peaked at 13 years, and possibly earlier among Black girls, which may be linked to an earlier start of menstruation.

American Diabetes Association guidelines recommend that clinicians screen overweight or obese youths for the disease starting at age 10 or once puberty starts, whichever is earlier, if they have one or more risk factors. These include a family history of the disease, signs of insulin resistance or affiliation with certain racial/ethnic groups, including Hispanic/Latino.

During checkups, clinicians can look for a visible sign of insulin resistance, an associated skin condition called acanthosis nigricans, says Paulina Cruz Bravo, a physician and diabetes researcher at Washington University School of Medicine in St. Louis. The skin changes tend to appear in the neck area or along folds in the skin, including in the armpits and on the elbows and knees, she says. “The top layer of the skin gets thickened. It’s described as a velvety appearance of the skin — it’s darker compared to the skin in other places.”

The thickened, darker, velvety skin shown here, known as acanthosis nigricans, is a potential warning sign of developing type 2 diabetes. The condition is likely to appear on the neck, elbows, knees and other areas where the skin folds. People who notice acanthosis nigricans on themselves or their children should bring it to a doctor’s attention. Credit: S. Dulebohn/Statpearls 2024

Where an adolescent carries any excess pounds also matters, as insulin resistance has been associated with a type of fat called visceral fat, says Alaina Vidmar, a pediatric endocrinologist at Children’s Hospital Los Angeles. Unlike the more common type of fat, called subcutaneous and felt by pinching around the waistline, visceral fat surrounds the liver and other vital organs, increasing the risk for type 2 diabetes, fatty liver disease and other conditions.

“You really need the liver to process glucose to be able to utilize your insulin well,” Vidmar says. “And if it is full of fat, you are unable to do that.” Fatty liver disease, which has been associated both with obesity and type 2 diabetes, is most common in Hispanic adults, followed by white adults and Black adults, according to a meta-analysis looking at 34 studies.

Imaging scans would be the ideal way to identify the extent and location of visceral fat in adolescents, Vidmar says. But given that routine scanning would be costly, clinicians can instead measure an adolescent’s waist circumference, “a great surrogate marker,” she says.

Diabetes risk depends not just on how much fat you carry, but where you carry it. People with an “apple” body shape, with much of their fat in the abdomen, are at higher risk of diabetes than those with a “pear” body shape, who carry their fat under the skin, especially on the hips.

Still, obesity accounts for only a portion of the type 2 risk profile, reflecting the complexities involved in understanding the pathophysiology of youth-onset disease. Roughly one-fourth of youths with type 2 diabetes are not obese, according to a meta-analysis published in 2022 in JAMA Network Open. Asian youths are least likely to be obese; roughly one-third don’t meet the criteria for obesity.

Moreover, while obesity and insulin resistance boost the risk of developing diabetes, those factors alone don’t predict whether an adolescent is eventually diagnosed with the disease, according to the authors of the Annual Review of Medicine overview. Instead, they point to the role of impaired beta cell function.

In one study involving 699 youths with type 2 diabetes, the standard antidiabetic drug metformin controlled blood glucose levels in only about half the participants. (The medication was least effective among Black youths, for reasons that are unclear, according to the researchers.) Another analysis of the same study population identified a 20 percent to 35 percent decline in beta function each year in diabetic youths, compared with prior studies showing about a 7 percent to 11 percent annual decline in diabetic adults.

“What we see in the youth is that beta cell function fails very rapidly,” Linder says, adding that the beta cell decline tends to correlate with the lack of response to metformin.

It’s unknown whether specific racial or ethnic groups are more vulnerable to loss of beta cell function, says Linder, who hopes that a new large-scale NIDDK study launching this summer will identify any such physiological and other differences among populations. The study, called Discovery of Risk Factors for Type 2 Diabetes in Youth Consortium, aims to enroll 3,600 overweight or obese adolescent boys and girls, 36 percent of them Hispanic. Bacha and other investigators on the project plan to follow the youths through puberty, looking at genetic and physiological markers such as insulin resistance and beta cell function. Their goal is to track who develops type 2 diabetes and what factors precipitate the disease.

In addition, researchers will learn about the participants’ mental health, lifestyles and social determinants of health, Linder says. To that end, families will be asked to share details about nutrition, physical activity and sleep, as well as food insecurity, exposure to racism and other stressors.

“Stress induces certain hormones that antagonize insulin, so they create more insulin resistance,” Linder says. “Stress also is associated with chronic inflammation in the body, which affects the ability of the body to respond normally.”

Young people experience many of the risk factors that predispose people to type 2 diabetes, such as prenatal exposures, junk food, sedentary lifestyles and high levels of stress.

Zooming in on Risk Factors in Hispanic Kids

Already, researchers who have studied at-risk Hispanic youths and their families have begun to flesh out environmental and other influences rooted in daily life that can boost the likelihood of obesity or diabetes. Michael Goran, a child obesity researcher at Children’s Hospital Los Angeles, has led a research project called the Study of Latino Adolescents at Risk (SOLAR), which tracked 328 Hispanic/Latino youths considered at highest risk of youth-onset diabetes based on their body mass index and family history of the disease. The participants, recruited in two waves between 2000 and 2015, completed health questionnaires and underwent annual exams, including imaging scans and other measurements.

One analysis found that Hispanic youths who lived in neighborhoods with higher levels of air pollution were more likely to experience a breakdown in beta cell function. “Which we weren’t necessarily expecting — we don’t know the mechanism of that,” says Goran, who coauthored a close look at pediatric insulin resistance in the 2005 Annual Review of Nutrition.

In more recent years, he’s turned his attention to studying nutrition shortly after birth, with a focus on infant formulas that contain corn syrup. Those formulas are more likely to spike blood sugar than are lactose-based formulas, he says. “If you’re spiking blood glucose with corn syrup in babies,” he says, “you can see how that would be problematic for long-term control of blood sugars.”

In one study, Goran and colleagues looked at obesity trends in 15,246 children who received formula through the Special Supplemental Nutrition Program for Women, Infants, and Children (WIC). Babies who consumed any formula with corn syrup were 10 percent more likely to be obese by age 2 than babies who didn’t. Nearly 90 percent of the study’s participants were Hispanic.

In other research, epidemiologist Carmen Isasi of the Albert Einstein College of Medicine in New York helped lead the Study of Latinos (SOL) Youth study, which delved into the extent to which a child’s family circumstances contribute to obesity and metabolic changes that may boost risk of youth-onset diabetes. Isasi and colleagues found chronic stress to be pervasive. Three-quarters of parents and caregivers reported stress and 29 percent detailed three or more stressors related to health, work or relationships. The higher the number of parental stressors, the more likely the child was to be obese.

Isasi also has looked at the relationship between food insecurity and metabolic health. Hispanic youths raised in households with the highest levels of food insecurity had significantly worse metabolic results, including elevated blood glucose and triglycerides, a type of cholesterol. Families dealing with food insecurity, Isasi says, probably have a lower-quality diet and skimp on costlier protein and fresh produce.

Preventing diabetes has proved challenging. A review paper looking at diet-related and other lifestyle initiatives targeting Hispanic youths found few studies to date that have shown improvements in body mass index or blood glucose levels.

Adolescents of lower socioeconomic status may also shoulder responsibilities that can undercut efforts to stay healthy, says Erica Soltero, a behavioral scientist at Houston’s Baylor College of Medicine, who works with Hispanic youths. For instance, older teens may struggle to attend an exercise class if they have an after-school job or must pick up younger siblings or start dinner. Technology, Soltero says, may be a better way to reach busy Hispanic teens; she’s piloting a study that will provide text-based lifestyle guidance to Hispanic teens with obesity.

Approved medication options remain limited for children and teens. If metformin doesn’t work, the alternative is insulin, and parents may resist giving injections because of the difficulties involved, Rodriguez says. She’s involved with an ongoing study in youths with type 2 diabetes to study the effectiveness of oral semaglutide, one of the newer diabetes drugs that also has achieved notable weight loss. Rodriguez estimates the results will be available by 2026.

The new NIDDK study won’t assess medication treatments, as it’s an observational study. But researchers involved are bullish that study-related insights could lead to better prevention and treatment approaches. “If someone is predisposed to beta cell dysfunction, should we be much more aggressive in treating their overweight/obesity,” Bacha says, “so that this beta cell function is preserved for a longer period of time?” Doctors could, for example, decide to start treatment earlier, she says.

Neither are researchers like Soltero deterred by the long-standing difficulties involved with revamping lifestyle habits. Soltero, who has worked with overweight and obese Hispanic adolescents to improve exercise and make dietary changes, describes them as often highly motivated given the damage they’ve seen the disease inflict on their own families.

“A lot of times they’ll have a touch point with a relative who’s on dialysis and maybe had a digit amputated,” Soltero says. Or “they’ll say, ‘I don’t want to prick myself every day like my Uncle So-and-So.’ Or ‘I don’t want to be on medicine for the rest of my life like my grandma.’ ”

[healixhospitals24]

Understand diabetes at 35: know diabetes, fight diabetes. Gain insights into managing diabetes effectively for a healthier lifestyle.

Do Visit: https://www.healixhospitals.com/blogs/diabetes-at-35:-know-diabetes.-fight-diabetes

[wellhealthhub]

11 Essential Facts About Diabetes Ketoacidosis (DKA) and How to Dodge It

Discover the essential facts about diabetes ketoacidosis (DKA) and how to avoid this serious complication of diabetes. Take control of your health and protect yourself. Introduction to Diabetes Ketoacidosis (DKA) Welcome to this comprehensive guide on Diabetes Ketoacidosis, commonly known as DKA. If you or a loved one is grappling with diabetes, understanding DKA is crucial. This…

[drcpanda12]

New Post has been published on https://www.knewtoday.net/unveiling-the-longest-recorded-coma-in-history-extraordinary-cases-of-prolonged-unconsciousness/

Unveiling the Longest Recorded Coma in History: Extraordinary Cases of Prolonged Unconsciousness

In medical science, a coma is a state of profound unconsciousness in which a person is unresponsive to external stimuli and cannot be awakened. It is a severe neurological condition that can be caused by various factors, such as traumatic brain injury, stroke, lack of oxygen to the brain, metabolic disorders, infections, or drug overdose.

During a coma, the person is unable to consciously perceive their surroundings, speak, or move purposefully. However, basic life-support functions such as breathing and circulation are usually preserved. The level and depth of coma can vary, with some individuals showing minimal brain activity and others displaying some limited responses.

Comas can last for a short period or extend for an indefinite duration, depending on the underlying cause and the individual’s response to treatment. Medical professionals assess coma patients using standardized scales, such as the Glasgow Coma Scale, to evaluate their level of consciousness and neurological function.

Medical professionals monitor and assess comatose patients closely using various diagnostic tests, such as brain imaging, electroencephalography (EEG), and neurological examinations, to determine the cause and potential prognosis. Treatment aims to address the underlying condition, provide supportive care, and promote recovery if possible. However, the outcome of a coma can vary widely, from full recovery to long-term disabilities or even death.

Treatment of coma focuses on addressing the underlying cause, providing supportive care, and preventing complications such as infections, pressure sores, or blood clots. In some cases, medications or surgery may be necessary to reduce swelling or treat the underlying condition. Rehabilitation is often required for individuals who emerge from a coma to regain lost physical and cognitive functions.

Causes of Coma

Drug Poisoning:

Responsible for 40% of comatose conditions. Some drugs, when used under particular conditions, can harm or decrease synaptic functioning in the ascending reticular activating system (ARAS), preventing the system from effectively arousing the brain.

Drug side effects such as irregular heart rate and blood pressure, as well as excessive breathing and perspiration, may also indirectly damage ARAS function and lead to coma. Because that drug poisoning is the cause of a high proportion of comas, hospitals screen all comatose patients by watching pupil size and eye movement via the vestibular-ocular reflex.

Cardiac Arrest:

Lack of oxygen, which usually results from cardiac arrest, is the second most prevalent cause of coma, accounting for around 25% of cases.

The Central Nervous System (CNS) relies heavily on oxygen to power its neurons. Hypoxia, or a lack of oxygen in the brain, causes sodium and calcium from outside the neurons to drop and intracellular calcium to rise, compromising neuron communication.

In the brain, a lack of oxygen induces ATP fatigue, cellular breakdown due to cytoskeleton damage, and nitric oxide generation.

Stroke-Related Coma

A stroke-related coma accounts for 20% of all comatose states. Blood flow to a portion of the brain is limited or blocked during a stroke.

Blood flow may be restricted as a result of an ischemic stroke, a brain hemorrhage, or a tumor. A lack of circulation to brain cells prevents oxygen from reaching the neurons, causing them to become disturbed and die. When brain cells die, brain tissue deteriorates, potentially impairing function.

Other Biological Conditions:

Trauma, severe blood loss, starvation, hypothermia, hyperthermia, hyperammonemia, aberrant glucose levels, and a variety of other biological conditions account for the remaining 15% of comatose patients. Additionally, studies reveal that 1 in every 8 patients with severe brain damage goes into a coma.

A coma scale is a mechanism for determining the degree of coma.

Glasgow Coma Scale

The Glasgow Coma Scale is a neurological scale that tries to provide a reliable, objective manner of monitoring a person’s conscious state for both initial and ongoing assessment. The criteria of the scale are applied to a patient, and the resulting points give a patient score ranging from 3(Three) indicating profound unconsciousness to 14 (Fourteen). GCS was originally designed to determine the degree of consciousness following a head injury, but it is now used by first responders, EMS, and clinicians to assess all acute medical and trauma patients. It is also used in hospitals for chronic patient monitoring, such as critical care.

 Longest Period of Time in Coma 

The longest recorded coma in medical history lasted for 37 years. The patient, Terry Wallis, was involved in a car accident in 1984 at the age of 19 and remained in a coma until 2003. During that time, he was unresponsive and completely dependent on medical care.

In 2003, Terry unexpectedly regained consciousness and started to communicate with his family. Although he was still severely disabled and had limited cognitive abilities, his recovery was considered remarkable given the length of time he spent in a coma.

It’s important to note that Terry Wallis’s case is exceptional and not representative of typical comas. Coma duration varies widely among individuals, and most comas are of much shorter duration. Medical professionals continue to research and study comes to better understand their causes and potential treatments.

Elaine Esposito :

According to Guinness World Records, he held the record for the longest duration of time in a coma, having lost consciousness in 1941 and died in that state more than 37 years later. Edwarda O’Bara and Aruna Shanbaug later broke Esposito’s record for the longest comas.

She was rushed to a hospital at the age of six with a burst appendix and underwent an appendectomy on August 6, 1941. She never regained consciousness after being sedated. She fell into convulsions as the procedure was drawing to a conclusion, her fever soared to 107.6 °F (42.0 °C), and physicians thought she would not survive the night. The origin of the issue was contested, with some claiming Elaine had encephalitis and others claiming her brain did not receive enough oxygen during the procedure.

Her parents spent the first 10 months of her coma in a Chicago hospital until they could no longer afford her treatment, at which time they moved her home so her mother Lucy could care for her 24 hours a day, seven days a week.

Throughout her extended coma, she had periods of both deep slumber and open-eyed unconsciousness, and she gained only a few pounds, reaching 85 pounds (39 kg). Elaine has overcome a variety of different health issues throughout the years, including more stomach surgery, pneumonia, measles, and a collapsed lung. The family subsequently relocated to Tarpon Springs, Florida, and she was also flown to Lourdes, France, to pray for a miracle.

 Elaine died at the age of 43 years and 357 days, having been in a coma for 37 years and 111 days.

Edwarda O’Bara :

After catching pneumonia in December 1969, he spent 42 years in a diabetic coma beginning in January 1970.

At the age of 16, O’Bara suffered pneumonia on December 20, 1969. Her condition deteriorated over the course of two weeks, and she was admitted to the hospital. According to her relatives, O’Bara “woke up shivering and in considerable pain because the oral type of insulin she had been taking wasn’t reaching her bloodstream” around 3 a.m. on January 3, 1970.

Her relatives hurried her to the hospital, where she succumbed to a diabetic coma. Edward begged her mother, Kaye O’Bara, not to leave her side before she slipped into a coma. She was fed by a tube, and Kaye repositioned her every two hours to prevent bedsores. Kaye also read to her, played music for her, and conversed with her. Joseph, her father, also quit his work to care for her. Kaye passed away in 2008, at the age of 81.

In conclusion, prolonged comas are rare and extraordinary medical conditions that challenge our understanding of the human brain and consciousness. The recorded cases of individuals who have spent extended periods in comas, such as Terry Wallis, Elaine Esposito, Edwarda O’Bara, and Sarah Scantlin, serve as remarkable examples of the resilience and unpredictability of the human body.

While the experience of individuals during a coma remains largely unknown, these cases highlight the potential for unexpected recoveries and the enduring dedication of caregivers. The medical community continues to explore the underlying causes and potential treatments for comas, seeking to improve our understanding and provide better care for affected individuals.

Though each coma case is unique, the stories of these individuals inspire hope and further our commitment to advancing medical research and support systems for those affected by coma. With ongoing research, continued advancements in medical technology, and dedicated healthcare professionals, we aim to improve outcomes and enhance the quality of life for individuals who experience prolonged comas.

Ultimately, the study of comas and their associated challenges fuels our collective pursuit of knowledge and pushes the boundaries of medical science, bringing us closer to unlocking the mysteries of consciousness and improving the lives of those affected by these profound states of unconsciousness.

[dostardoy]

I either eat too little or like a fat f*ck. There is no in-between

[willowreader]

Excellent article from a scientist who does great research and has her own lab.

[strv2bepretty]

New diet plan

DIET - OMAD/intermittent fasting (choose one of 2 meals)

Eat in front of people so they don't get suspicous

Breakfast -

Options:

protein shake

fruit

eggs

milk

veggies

Dinner -

Options:

protein shake smoothie

eggs

salad

fruit

soup

(max: 600 cals allowed per day, and 700-800 on re-feeding times when metabolism gets slow)

(allowed to: drink black coffee & tea, chew gum)

Workouts (do all)

5 min warmup (no jumping) - MadFit

Get abs in 2 weeks - Chloe Ting

10 min break

Pilates workout at home - Shirlyn Kim

10 min break

9 mins thigh & legs workout - Shirlyn Kim

1 hour break

walk 30 mins - 1 hr

MOST IMPORTANT - DRINK 64 fl oz OF WATER EVERYDAY

[sk1n4ndb0ne]

Metabolism days actually work ... I was hitting a plateau, so I decided to eat normally over the weekend ... and I lost weight. Feeling super happy right now.

[painted-bees]

So Haze found an eldritch kin who--despite being a far less lazy shifter than Cortes, isn't fooling me for a second.

Her name is Arrow De Wilde and she's my new reference model for Cortes shapes and poses lmao

Look at her in motion:

It's almost unreal. She's incredible.

[donuteet]

HAVING TO FORCE MYSELF TO EAT MORE IS SO SCARYy

[fa3zb0nez]

it’s a metab day it’s a metab day it’s a metab day

[athetos]

A woman at work asked if the packages I were carrying were heavy, and considering I was pretty easily balancing all 3 in one hand, I said no, but she took the larger one with both hands and said “oh, this one is heavy!” And I blinked at her in befuddlement and then assured her the other two were very light and I’d trade with her. I was thinking afterwards “wow, I know she was slim and barely 5 feet but I could barely tell there was anything in them” then I remembered when I was home earlier in the month and my family kept commenting on my arms and my mom was struggling with a huge bag of dog food so I took it and threw it on my shoulder and climbed the stairs and she told me I’m getting too buff…

[healixhospitals24]

Diabetes At 35: Know Diabetes. Fight Diabetes

At 35, life is often at its peak - careers are thriving, families are growing, and adventures await. However, amidst this hustle and bustle, it's crucial to pay attention to our health. One such health concern that can sneak up on us, particularly at this age, is diabetes. In this blog, we'll delve into what diabetes at 35 entails, why it's important to be aware, and how to effectively combat it.

Diabetes: The Silent Intruder

At 35, life often feels like it's just beginning. However, this milestone also marks a crucial time to assess your health, especially concerning diabetes. Here's a breakdown of what you need to know:

Types of Diabetes: Understand the differences between Type 1 and Type 2 diabetes, along with lesser-known types like gestational diabetes.

Risk Factors: Explore the various risk factors that predispose individuals to diabetes, including genetics, lifestyle choices, and medical history.

Symptoms: Recognizing the early signs of diabetes can empower you to take action before it escalates. Symptoms such as frequent urination, increased thirst, and unexplained weight loss should not be overlooked.

What are the differences between type 1 and type 2 diabetes treatment options?

The differences between type 1 and type 2 diabetes treatment options are as follows:

Type 1 Diabetes:

Treatment: Requires insulin replacement therapy as the pancreas does not produce insulin.

Management: Lifelong insulin therapy is essential for individuals with type 1 diabetes.

Complications: Without insulin, individuals with type 1 diabetes are at risk of developing life-threatening conditions like diabetic ketoacidosis.

Type 2 Diabetes:

Treatment: Can be managed with lifestyle changes, diet, weight loss, medications, and sometimes insulin.

Prevention: Lifestyle modifications can help prevent or reduce symptoms of type 2 diabetes.

Complications: Individuals with type 2 diabetes may develop hyperosmolar coma due to high blood sugar levels.

Key Differences:

Insulin Dependency: Type 1 diabetes requires insulin for survival, while type 2 diabetes may not initially need insulin.

Onset and Symptoms: Type 1 diabetes often presents in childhood or adolescence with sudden symptoms, while type 2 diabetes is more common in older adults and symptoms may be vague.

Risk Factors: Type 1 diabetes is primarily genetic, while type 2 diabetes is influenced by lifestyle factors like obesity, sedentary lifestyle, and diet.

It's crucial for individuals with either type of diabetes to closely follow up with healthcare providers to manage their condition effectively and prevent complications

What are the common symptoms of diabetes at 35?

Common symptoms of diabetes at 35 can include:

Increased thirst

Frequent urination

Increased hunger

Unintended weight loss

Fatigue

Blurred vision

Slow-healing sores

Numbness or tingling in the hands or feet

Areas of darkened skin, usually in the armpits and neck

These symptoms may not always be noticeable until blood sugar levels are significantly high. It is essential to be aware of these signs and seek medical attention if you experience them, especially if you are 35 years old and at risk of diabetes. for a brighter, healthier future.

Read More: https://www.healixhospitals.com/blogs/diabetes-at-35:-know-diabetes.-fight-diabetes

[wellhealthhub]

"Get Rid of Diabetes Fast: The 7-Day Miracle Plan"

Are you tired of living with diabetes? Are you ready to take control of your health and say goodbye to this condition for good? Look no further! We have the perfect solution for you—a 7-day miracle plan that will transform your life and help you reclaim your health. Get ready to bid farewell to diabetes and embark on a journey towards a happier, healthier you! Say Goodbye to Diabetes: Unveiling…

[notskinnyleeyuh]

always blaming your body on a slow metabolism is chill until you realize how different skinny peoples habits are.

[princessjulietskingdom]

Ik u already spoke about reverse diet but can u go more into detail pls, im worried about gaining, is this metabolism thing meant to happen every week or every two weeks? I dont like the thought of it being every week bc just gaining sounds awful after my progress

Hi love! Reverse diet is actually not that scary thing as we used to think. You can start on your current cal limit and add 50-100 cals to that number. At first few days it might seem that you start to gain weight (1-3 pounds) but as you continue with that cal limit the "extra" pounds will drop and thats when you know your body is adapting to that new cal limit. You can eat that way 1 to 7 more days and then start rising your cal intake for other 50-100 cal. So i'd say you only need to do that every 2 to 3 weeks. What makes you comfortable. If gaining temporaly pounds is scaring you, you can also increase your activity level. But you have to make sure that you eat the burned cals. That way you can kick start your metabolism to life again. I hope this helped even for a little.